Forsythiaside inhibits bacterial adhesion on titanium alloy and attenuates Ti-induced activation of nuclear factor-κB signaling-mediated macrophage inflammation.

Background: Inflammation and biofilm formation by Staphylococcus aureus (S. aureus) are common causes of periprosthetic infection and loosening. Recently, we identified that forsythiaside is bacteriostatic for S. aureus and methicillin-resistant S. aureus (MRSA). The purpose of the present study was...

Descripción completa

Detalles Bibliográficos
Publicado en:Journal of Orthopaedic Surgery & Research Vol. 13; no. 1; pp. 1 - 11
Autores principales: Haifeng Li, Dongmei Tang, Chao Qi, Xia Zhao, Guangchao Wang, Yi Zhang, Tengbo Yu
Formato: pictorial research tables/charts Journal Article
Publicado: BioMed Central 6/5/2018
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=130238021&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 130238021
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        1749799X
        38N9
      jtl: Journal of Orthopaedic Surgery & Research
      issn: 1749799X
      maglogo: N
    pubinfo:
      dt: 6/5/2018
      vid: 13
      iid: 1
      pid: 24147
      pub: BioMed Central
    artinfo:
      ui:
        130238021
        130238021
        130238021
        10.1186/s13018-018-0834-x
        130238021
      ppf: 1
      ppct: 10
      formats:
      tig:
        atl: Forsythiaside inhibits bacterial adhesion on titanium alloy and attenuates Ti-induced activation of nuclear factor-κB signaling-mediated macrophage inflammation.
      aug:
        au:
          Haifeng Li
          Dongmei Tang
          Chao Qi
          Xia Zhao
          Guangchao Wang
          Yi Zhang
          Tengbo Yu
        affil: Department of Sports Medicine, The Affiliated Hospital of Qingdao University, No. 16 Jiangsu Road, Qingdao 266003, China
      sug:
        subj:
          Plant Extracts Pharmacodynamics
          Bacterial Physiology Drug Effects
          NF-kappa B Drug Effects
          Signal Transduction Drug Effects
          Macrophages Drug Effects
          Inflammation Prevention and Control
          Biofilms Drug Effects
          Human
          In Vitro Studies
          Microscopy Methods
          Microscopy, Electron, Scanning
          Reverse Transcriptase Polymerase Chain Reaction Methods
          Blotting, Western
          Colony Count, Microbial
          Gene Expression Drug Effects
          Cytokines Drug Effects
          Staphylococcus Aureus Drug Effects
          Methicillin-Resistant Staphylococcus Aureus Drug Effects
          Joint Prosthesis
          Orthopedic Prosthesis
          Titanium Adverse Effects
      ab: Background: Inflammation and biofilm formation by Staphylococcus aureus (S. aureus) are common causes of periprosthetic infection and loosening. Recently, we identified that forsythiaside is bacteriostatic for S. aureus and methicillin-resistant S. aureus (MRSA). The purpose of the present study was to examine the effect of forsythiaside on S. aureus and MRSA adhesion and biofilm formation on the surface of titanium alloy, which is a popular material for orthopedic joint prostheses. Methods: Two strains of S. aureus and MRSA were used for in vitro experiments. The spread plate method, confocal laser scanning microscopy (CLSM), and scanning electron microscopy (SEM) were used to characterize antimicrobial activity of forsythiaside. Real-time polymerase chain reaction (RT-PCR) and western blotting were used to investigate the inhibitory level of forsythiaside required for titanium-associated inflammation. Results: Direct colony counting showed that 16 μg/mL forsythiaside significantly inhibited S. aureus and MRSA adhesion on titanium alloy discs in 2 h. CLSM and SEM showed that higher concentrations (> 30 mg/mL) of forsythiaside effectively inhibited the adhesion of S. aureus and MRSA on the surface of the titanium disc in 24 h. Forsythiaside was capable of attenuating Ti-induced activation of nuclear factor-κB signaling, targeting IxB kinase-α (IKKα) kinases of macrophages, and influencing the expression of NF-κB downstream cytokines. Conclusions: These observations suggest that forsythiaside is a potential agent for the treatment of Ti implant- associated infection and inflammation.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N