Analysis of VSX1 variations in Brazilian subjects with keratoconus.

Purpose: To screen visual system homeobox 1 (VSX1) gene in Brazilian subjects affected with keratoconus (KCN). Methods: Seventy-three patients with KCN and 106 healthy controls were enrolled in this study. Patients were diagnosed with KCN based on eye examination and corneal topographic features acc...

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Publicado en:Journal of Ophthalmic & Vision Research Vol. 13; no. 3; pp. 266 - 274
Autores principales: Silva, Dulceria, Gadelha, Bianca, Feitosa, Alex, Silva, Rafaela, Albuquerque, Tarsila, Santos, Débora, Gadelha, Diego, Fonseca Schamber-Reis, Bruno
Formato: pictorial research tables/charts Journal Article
Publicado: Knowledge E DMCC Jul-Sep2018
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jul-Sep2018
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      pub: Knowledge E DMCC
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        atl: Analysis of VSX1 variations in Brazilian subjects with keratoconus.
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        au:
          Silva, Dulceria
          Gadelha, Bianca
          Feitosa, Alex
          Silva, Rafaela
          Albuquerque, Tarsila
          Santos, Débora
          Gadelha, Diego
          Fonseca Schamber-Reis, Bruno
        affil: Department of Medical Genetics, School of Medical Sciences, UNIFACISA, Campina Grande; Federal University of Paraíba, Campina Grande, Paraíba
      sug:
        subj:
          Keratoconus Diagnosis
          Genetic Screening
          Polymorphism, Genetic
          Human
          Brazil
          Diagnosis, Eye
          Corneal Topography
          Blood Donors
          Amino Acids
          DNA
          Genotype
          Mutation
          Silicon Analysis
          Severity of Illness
          Adolescence
          Child
          Child, Preschool
          Infant
          Adolescent: 13-18 years
          Child: 6-12 years
          Child, Preschool: 2-5 years
          Infant: 1-23 months
      ab: Purpose: To screen visual system homeobox 1 (VSX1) gene in Brazilian subjects affected with keratoconus (KCN). Methods: Seventy-three patients with KCN and 106 healthy controls were enrolled in this study. Patients were diagnosed with KCN based on eye examination and corneal topographic features according to Rabinowitz's criteria (K > 47.2, I-S > 1.4 and KISA > 100%). DNA from blood samples was extracted from donors, and the exons and exon-intron boundaries of VSX1 were sequenced. The potential impact of the identified amino acid changes was assessed with Poly-Phen2, SIFT, and PMUT analysis tools. Genotyping was confirmed by RLFP technique, which was also applied to genotype non-affected individuals. Results: We found three non-synonymous substitutions (L68H, R131S, and D105E) in VSX1 exon 1, with L68H mutation as a novel variation in this gene. In silico analysis indicated that all variations found were predicted to be probably damaging to VSX1 structure and function. Examination of R131S and L68H variations segregating in one family suggested a strong effect of these variations in increasing disease severity in the proband, which presented bilateral KCN leading to corneal grafting before the age of sixteen. We found a novel synonymous substitution (P79P) and two previously described exonic polymorphisms, with unknown roles in VSX1 pathogenesis. Conclusion:VSX1 polymorphisms found in the Brazilian population support a genetic component in KCN pathogenesis. L68H is a novel mutation, and the phenotypic data suggest that this mutation might enhance disease severity when combined with other polymorphisms. However, further investigations are needed.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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