Targeting autophagy by small molecule inhibitors of vacuolar protein sorting 34 (Vps34) improves the sensitivity of breast cancer cells to Sunitinib.

Resistance to chemotherapy is a challenging problem for treatment of cancer patients and autophagy has been shown to mediate development of resistance. In this study we systematically screened a library of 306 known anti-cancer drugs for their ability to induce autophagy using a cell-based assay. 11...

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Publicado en:Cancer Letters Vol. 435; pp. 32 - 44
Autores principales: Dyczynski, Matheus, Yu, Yasmin, Otrocka, Magdalena, Parpal, Santiago, Braga, Tiago, Henley, Aine Brigette, Zazzi, Henric, Lerner, Mikael, Wennerberg, Krister, Viklund, Jenny, Martinsson, Jessica, Grandér, Dan, De Milito, Angelo, Pokrovskaja Tamm, Katja
Formato: research Journal Article
Publicado: Elsevier B.V. Oct2018
Acceso en línea:Ver este registro en EBSCOhost
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        03043835
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      jtl: Cancer Letters
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      dt: Oct2018
      vid: 435
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      pub: Elsevier B.V.
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        10.1016/j.canlet.2018.07.028
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        atl: Targeting autophagy by small molecule inhibitors of vacuolar protein sorting 34 (Vps34) improves the sensitivity of breast cancer cells to Sunitinib.
      aug:
        au:
          Dyczynski, Matheus
          Yu, Yasmin
          Otrocka, Magdalena
          Parpal, Santiago
          Braga, Tiago
          Henley, Aine Brigette
          Zazzi, Henric
          Lerner, Mikael
          Wennerberg, Krister
          Viklund, Jenny
          Martinsson, Jessica
          Grandér, Dan
          De Milito, Angelo
          Pokrovskaja Tamm, Katja
        affil: Department of Oncology-Pathology, Cancer Center Karolinska, Karolinska Institutet, Stockholm, Sweden
      sug:
        subj:
          Autophagy Drug Effects
          Laboratory Chemicals Pharmacodynamics
          Breast Neoplasms Drug Therapy
          Phosphotransferases Antagonists and Inhibitors
          Breast Neoplasms Metabolism
          Phosphotransferases Metabolism
          Human
          Mice
          Protein Kinase Inhibitors Pharmacodynamics
          Cell Line, Tumor
          Drug Screening Assays, Antitumor Methods
          Cell Physiology Drug Effects
          Apoptosis Drug Effects
          Animal Studies
          Breast Neoplasms Pathology
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
      ab: Resistance to chemotherapy is a challenging problem for treatment of cancer patients and autophagy has been shown to mediate development of resistance. In this study we systematically screened a library of 306 known anti-cancer drugs for their ability to induce autophagy using a cell-based assay. 114 of the drugs were classified as autophagy inducers; for 16 drugs, the cytotoxicity was potentiated by siRNA-mediated knock-down of Atg7 and Vps34. These drugs were further evaluated in breast cancer cell lines for autophagy induction, and two tyrosine kinase inhibitors, Sunitinib and Erlotinib, were selected for further studies. For the pharmacological inhibition of autophagy, we have characterized here a novel highly potent selective inhibitor of Vps34, SB02024. SB02024 blocked autophagy in vitro and reduced xenograft growth of two breast cancer cell lines, MDA-MB-231 and MCF-7, in vivo. Vps34 inhibitor significantly potentiated cytotoxicity of Sunitinib and Erlotinib in MCF-7 and MDA-MB-231 in vitro in monolayer cultures and when grown as multicellular spheroids. Our data suggests that inhibition of autophagy significantly improves sensitivity to Sunitinib and Erlotinib and that Vps34 is a promising therapeutic target for combination strategies in breast cancer.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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