CRISPR-Cas9 Mediated RNase L Knockout Regulates Cellular Function of PK-15 Cells and Increases PRV Replication.

Ribonuclease L (RNase L) is an important antiviral endoribonuclease regulated by type I IFN. RNase L is activated by viral infection and dsRNA. Because the role of swine RNase L (sRNase L) is not fully understood, in this study, we generated a sRNase L knockout PK-15 (KO-PK) cell line through the CR...

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Publicado en:BioMed Research International pp. 1 - 11
Autores principales: Sui, Chao, Jiang, Dandan, Wu, Xiangju, Cong, Xiaoyan, Li, Feng, Shang, Yingli, Wang, Jinqiu, Liu, Sidang, Shan, Hu, Qi, Jing, Du, Yijun
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 3/3/2019
Acceso en línea:Ver este registro en EBSCOhost
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    pubinfo:
      dt: 3/3/2019
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        135031900
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        10.1155/2019/7398208
        135031900
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        atl: CRISPR-Cas9 Mediated RNase L Knockout Regulates Cellular Function of PK-15 Cells and Increases PRV Replication.
      aug:
        au:
          Sui, Chao
          Jiang, Dandan
          Wu, Xiangju
          Cong, Xiaoyan
          Li, Feng
          Shang, Yingli
          Wang, Jinqiu
          Liu, Sidang
          Shan, Hu
          Qi, Jing
          Du, Yijun
        affil: Shandong Provincial Key Laboratory of Animal Biotechnology and Disease Control and Prevention, College of Animal Science and Technology, Shandong Agricultural University, Tai'an 271018, China
      sug:
        subj:
          Herpesviruses Metabolism
          Epithelial Cells Physiology
          CRISPR-Associated Proteins
          Genetic Techniques
          Purine Nucleosides
          Electrophoresis, Agar Gel
          Sequence Analysis Methods
          Blotting, Western
          Apoptosis
          Herpesviruses Drug Effects
          CRISPR-Associated Proteins Pharmacodynamics
          Interferons
          Animal Studies
          Swine
          RNA, Messenger
      ab: Ribonuclease L (RNase L) is an important antiviral endoribonuclease regulated by type I IFN. RNase L is activated by viral infection and dsRNA. Because the role of swine RNase L (sRNase L) is not fully understood, in this study, we generated a sRNase L knockout PK-15 (KO-PK) cell line through the CRISPR/Cas9 gene editing system to evaluate the function of sRNase L. After transfection with CRISPR-Cas9 followed by selection using puromycin, sRNase L knockout in PK-15 cells was further validated by agarose gel electrophoresis, DNA sequencing, and Western blotting. The sRNase L KO-PK cells failed to trigger RNA degradation and induced less apoptosis than the parental PK-15 cells after transfected with poly (I: C). Furthermore, the levels of ISGs mRNA in sRNase L KO-PK cells were higher than those in the parental PK-15 cells after treated with poly (I: C). Finally, both wild type and attenuated pseudorabies viruses (PRV) replicated more efficiently in sRNase L KO-PK cells than the parental PK-15 cells. Taken together, these findings suggest that sRNase L has multiple biological functions including cellular single-stranded RNA degradation, induction of apoptosis, downregulation of transcript levels of ISGs, and antiviral activity against PRV. The sRNase L KO-PK cell line will be a valuable tool for studying functions of sRNase L as well as for producing PRV attenuated vaccine.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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