Differential Potentiation of Retinoic Acid Effects against Human Breast Cancer Cells by Unsaturated Fatty Acids.

Retinoic acid (RA) and unsaturated fatty acids (UFA) are proposed as nutritional anticancer agents. Nonetheless, the activity of their combination on human breast cancer needs further study. Our aim was to evaluate this activity on the MCF-7 and ZR-75-1 cell lines treated with 1 µM RA and 50 µM of γ...

Descripción completa

Detalles Bibliográficos
Publicado en:Nutrition & Cancer Vol. 70; no. 7; pp. 1137 - 1145
Autores principales: Quiroga, Patricia L., Soria, Elio A., Valentich, Mirta A., Eynard, Aldo R.
Formato: research Journal Article
Publicado: Taylor & Francis Ltd Oct2018
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=135476388&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 135476388
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        01635581
        7MS
      jtl: Nutrition & Cancer
      issn: 01635581
      maglogo: N
    pubinfo:
      dt: Oct2018
      vid: 70
      iid: 7
      pid: 377
      pub: Taylor & Francis Ltd
      place: Philadelphia, Pennsylvania
    artinfo:
      ui:
        135476388
        135476388
        135476388
        10.1080/01635581.2018.1497669
        135476388
      ppf: 1137
      ppct: 8
      formats:
        fmt:
          – @attributes:
              type: T
          – @attributes:
              type: P
      tig:
        atl: Differential Potentiation of Retinoic Acid Effects against Human Breast Cancer Cells by Unsaturated Fatty Acids.
      aug:
        au:
          Quiroga, Patricia L.
          Soria, Elio A.
          Valentich, Mirta A.
          Eynard, Aldo R.
        affil: Cátedra de Biología Celular, Histología y Embriología, Instituto de Biología Celular, Facultad de Ciencias Médicas, Universidad Nacional de Córdoba, Córdoba, Argentina
      sug:
        subj:
          Breast Neoplasms Drug Therapy
          Fatty Acids, Unsaturated Pharmacodynamics
          Tretinoin Pharmacodynamics
          Tretinoin Administration and Dosage
          Fatty Acids, Unsaturated Administration and Dosage
          Cell Line, Tumor Drug Effects
          Drug Synergism
          Human
          Eicosapentaenoic Acid Pharmacodynamics
          Eicosanoids Pharmacodynamics
          Linoleic Acids Pharmacodynamics
          Analysis of Variance
          Fisher's Exact Test
          Regression
          Cell Differentiation Drug Effects
          Cell Viability Drug Effects
          gamma-Glutamyltransferase Drug Effects
          Cell Adhesion Molecules Drug Effects
          Microfilament Proteins Drug Effects
      ab: Retinoic acid (RA) and unsaturated fatty acids (UFA) are proposed as nutritional anticancer agents. Nonetheless, the activity of their combination on human breast cancer needs further study. Our aim was to evaluate this activity on the MCF-7 and ZR-75-1 cell lines treated with 1 µM RA and 50 µM of γ-linoleic (GLA, ω-6), eicosapentaenoic (EPA, ω-3), oleic (OA, ω-9), or eicosatrienoic (ETA, ω-9) acids. The following cellular responses were compared by ANOVA and Fisher test (P < 0.05): fatty acids, E-cadherin, actin (differentiation), conjugated dienes, γ-glutamyltranspeptidase activity (stress), and viability, which were correlated by partial least squares regression. Although both cell lines responded differentially, RA modified unsaturated fatty acids, increased differentiation, reduced γ-glutamyltranspeptidase, and viability. RA differentiating activity on ZR-75-1 was morphologically enhanced by UFA. Stress induction with γ-glutamyltranspeptidase decrease and conjugated dienes was promoted by ETA in MCF-7, and EPA and OA in ZR-75-1. RA-related reduced viability was potentiated by EPA and OA in both lines. GLA was less active. Therefore, unsaturated fatty acids (ω-3/ω-9) potentiated the multitarget retinoic acid activity against these human breast cancer cells.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N