Lipoprotein(a) Particle Production as a Determinant of Plasma Lipoprotein(a) Concentration Across Varying Apolipoprotein(a) Isoform Sizes and Background Cholesterol-Lowering Therapy.

Background Elevated lipoprotein(a) (Lp(a)), a low-density lipoprotein-like particle bound to the polymorphic apolipoprotein(a) (apo(a)), may be causal for cardiovascular disease. However, the metabolism of Lp(a) in humans is poorly understood. Methods and Results We investigated the kinetics of Lp(a...

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Publicado en:Journal of the American Heart Association Vol. 8; no. 7; pp. 1 - 13
Autores principales: Chan, Dick C., Watts, Gerald F., Coll, Blai, Wasserman, Scott M., Marcovina, Santica M., Barrett, P. Hugh R.
Formato: research Journal Article
Publicado: Wiley-Blackwell 4/2/2019
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 4/2/2019
      vid: 8
      iid: 7
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1161/JAHA.118.011781
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        135675983
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        atl: Lipoprotein(a) Particle Production as a Determinant of Plasma Lipoprotein(a) Concentration Across Varying Apolipoprotein(a) Isoform Sizes and Background Cholesterol-Lowering Therapy.
      aug:
        au:
          Chan, Dick C.
          Watts, Gerald F.
          Coll, Blai
          Wasserman, Scott M.
          Marcovina, Santica M.
          Barrett, P. Hugh R.
        affil: Schools of Medicine University of Western Australia, Perth, Australia
      sug:
        subj:
          Apolipoproteins
          Lipoproteins, LDL Cholesterol Metabolism
          Male
          Adult
          Lipoproteins, HDL Cholesterol Metabolism
          Human
          Young Adult
          Hypercholesterolemia, Familial Blood
          Antilipemic Agents Therapeutic Use
          Lipoproteins Metabolism
          Adolescence
          Proteins
          Lipoproteins Blood
          Hypercholesterolemia, Familial Prevention and Control
          Aged
          Middle Age
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
          Scales
          Adult: 19-44 years
          Adolescent: 13-18 years
          Aged: 65+ years
          Middle Aged: 45-64 years
          Male
      ab: Background Elevated lipoprotein(a) (Lp(a)), a low-density lipoprotein-like particle bound to the polymorphic apolipoprotein(a) (apo(a)), may be causal for cardiovascular disease. However, the metabolism of Lp(a) in humans is poorly understood. Methods and Results We investigated the kinetics of Lp(a)-apo(a) and low-density lipoprotein-apoB-100 in 63 normolipidemic men. The fractional catabolic rate ( FCR ) and production rate PR ) were studied. Plasma apo(a) concentration was significantly and inversely associated with apo(a) isoform size ( r=-0.536, P<0.001) and apo(a) FCR ( r=-0.363, P<0.01), and positively with apo(a) PR ( r=0.877, P<0.001). There were no significant associations between the FCR s of apo(a) and low-density lipoprotein-apoB-100. Subjects with smaller apo(a) isoform sizes (≤22 kringle IV repeats) had significantly higher apo(a) PR ( P<0.05) and lower apo(a) FCR ( P<0.01) than those with larger sizes. Plasma apo(a) concentration was significantly associated with apo(a) PR ( r=0.930, P<0.001), but not with FCR ( r=-0.012, P>0.05) in subjects with smaller apo(a) isoform size. In contrast, both apo(a) PR and FCR were significantly associated with plasma apo(a) concentrations ( r=0.744 and -0.389, respectively, P<0.05) in subjects with larger isoforms. In multiple regression analysis, apo(a) PR and apo(a) isoform size were significant predictors of plasma apo(a) concentration independent of low-density lipoprotein-apoB-100 FCR and background therapy with atorvastatin and evolocumab. Conclusions In normolipidemic men, the plasma Lp(a) concentration is predominantly determined by the rate of production of Lp(a) particles, irrespective of apo(a) isoform size and background therapy with a statin and a proprotein convertase subtilisin-kexin type 9 inhibitor. Our findings underscore the importance of therapeutic targeting of the hepatic synthesis and secretion of Lp(a) particles. Lp(a) particle catabolism may only play a modest role in determining Lp(a) concentration in subjects with larger apo(a) isoform size. Clinical Trial Registration URL : http://www.clinicaltrials.gov . Unique identifier: NCT 02189837.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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