Electrical Stimulation Activates Fibroblasts through the Elevation of Intracellular Free Ca2+: Potential Mechanism of Pelvic Electrical Stimulation Therapy.

Ca2+ is an important ion in response to electrical stimulation (ES) and acts as second messenger in the regulation of various physiological processes. Pelvic floor electrical stimulation (PES) is a low-voltage clinical application, available for urinary incontinence (UI) treatment. Fibroblasts, as t...

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Publicado en:BioMed Research International pp. 1 - 11
Autores principales: Li, Suting, Lu, Danhua, Tang, Jianming, Min, Jie, Hu, Ming, Li, Yang, Liu, Yaodan, Wang, Linlin, Liu, Cheng, Hong, Li
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 4/21/2019
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 4/21/2019
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        135973138
        10.1155/2019/7387803
        135973138
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        atl: Electrical Stimulation Activates Fibroblasts through the Elevation of Intracellular Free Ca2+: Potential Mechanism of Pelvic Electrical Stimulation Therapy.
      aug:
        au:
          Li, Suting
          Lu, Danhua
          Tang, Jianming
          Min, Jie
          Hu, Ming
          Li, Yang
          Liu, Yaodan
          Wang, Linlin
          Liu, Cheng
          Hong, Li
        affil: Department of Gynecology and Obstetrics, Renmin Hospital of Wuhan University, Wuhan, Hubei, China
      sug:
        subj:
          Urinary Incontinence Therapy
          Electric Stimulation Methods
          Pelvic Floor Muscles
          Fibroblasts
          Calcium
          Animal Studies
          Mice
          Cell Proliferation
          Phosphorylation
          Carrier Proteins
          Treatment Outcomes
      ab: Ca2+ is an important ion in response to electrical stimulation (ES) and acts as second messenger in the regulation of various physiological processes. Pelvic floor electrical stimulation (PES) is a low-voltage clinical application, available for urinary incontinence (UI) treatment. Fibroblasts, as the main cellular component of vaginal wall and pelvic ligament, play an important role in the maintenance of pelvic health. We studied the effect of ES on fibroblasts in this study. ES was conducted with electrotaxis chambers on L929 fibroblast and the ES parameter was 100 mV/mm×2h. The results showed that ES increased intracellular Ca2+ concentration, promoted the expression of PCNA, CyclinB1, and CyclinD1, and increased the proportion of cells in S and G2 phages. After ES, fibroblasts get activated and proliferated. Besides, BAPTA-AM, a membrane permeated chelator for intracellular free Ca2+, partially inhibited the effect of ES on fibroblasts activation and proliferation promotion. Furthermore, we elucidated that Ca2+, as a second messenger and upstream signal for Smads and Akt signaling, regulated ES-induced nuclear translocation of smad2/3, phosphorylation of smad2/3, Akt, and GSK3β. Finally, we validated the effect of ES on PES mouse model. The results indicated that PES promoted the activation and proliferation of fibroblasts in vivo. In conclusion, we verify that ES can elevate the concentration of intracellular Ca2+ and activate its downstream signaling and then promote the activation of fibroblasts, which may be one of the mechanisms of PES therapy.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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