Gossypol Promotes Wnt/β-Catenin Signaling through WIF1 in Ovariectomy-Induced Osteoporosis.

Osteoporosis is one of the most frequent diseases related with age. Previously, we have reported a novel potential drug, gossypol, for the treatment of osteoporosis through its regulation of Wnt/β-catenin signaling. This study aims to identify the detailed mechanism of gossypol in human osteoporosis...

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Publicado en:BioMed Research International pp. 1 - 11
Autores principales: Liang, Jinqian, Chen, Chong, Liu, Hongzhe, Liu, Xiangyang, Zhao, Hong, Hu, Jianhua
Formato: research Journal Article
Publicado: Wiley-Blackwell 4/21/2019
Acceso en línea:Ver este registro en EBSCOhost
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      issn: 23146133
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      dt: 4/21/2019
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        135973140
        135973140
        135973140
        10.1155/2019/8745487
        135973140
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        atl: Gossypol Promotes Wnt/β-Catenin Signaling through WIF1 in Ovariectomy-Induced Osteoporosis.
      aug:
        au:
          Liang, Jinqian
          Chen, Chong
          Liu, Hongzhe
          Liu, Xiangyang
          Zhao, Hong
          Hu, Jianhua
        affil: Department of Orthopaedic Surgery, Peking Union Medical College Hospital, Beijing 100730, China
      sug:
        subj:
          Contraceptive Agents, Male Pharmacodynamics
          Oophorectomy Adverse Effects
          Osteoporosis Etiology
          Osteoporosis Drug Therapy
          Signal Transduction Drug Effects
          Cytoskeletal Proteins Therapeutic Use
          Human
          Wnt Proteins Drug Effects
          Animal Studies
          Mice
          RNA
          Sequence Analysis
          Polymerase Chain Reaction Methods
          Blotting, Western
          Biological Assay Methods
          Cell Viability Evaluation
          Cell Proliferation Drug Effects
          Apoptosis Drug Effects
          Models, Biological
      ab: Osteoporosis is one of the most frequent diseases related with age. Previously, we have reported a novel potential drug, gossypol, for the treatment of osteoporosis through its regulation of Wnt/β-catenin signaling. This study aims to identify the detailed mechanism of gossypol in human osteoporosis. Mice injected with gossypol were subjected for RNA-seq analysis and the transcription level of WIF1 was shown to be decreased dramatically in gossypol-treated mice, which was further confirmed by qRT-PCR and western blot analysis. Luciferase reporter assay showed gossypol inhibited the activity of WIF1 and the methylation of WIF1 was significantly upregulated, evidenced by ChIP assay. Cell viability assays demonstrated that gossypol promoted cell proliferation while cotreatment with WIF1 expressing plasmid reversed the effect in a dose- and time-dependent manner. Similarly, cell apoptotic assays and TUNEL assays showed gossypol suppressed cell apoptosis, which was revised by WIF1 overexpression. The mouse model suggested gossypol injection ameliorated osteoporosis, while coinjection of AAV5-WIF1 eliminated the protection effects of gossypol, as evidenced by H&E staining, serum osteocalcin level, serum OPG level, serum RANKL level, bone density, ultimate strength, and postyield displacement. This study is a supplement to the former publication, which reinforced the protection effect of gossypol in human osteoporosis.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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