Thalidomide-induced neuropathy.

Background: Thalidomide is effective for the treatment of some refractory dermatologic and oncologic diseases. Toxic neuropathy limits its use, as embryopathy can be avoided by contraceptive measures.Objective: To describe the clinical, electrophysiologic, and pathologic features of thalidomide-indu...

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Publicado en:Neurology Vol. 59; no. 12; pp. 1872 - 1876
Autores principales: Chaudhry, V, Cornblath, D R, Corse, A, Freimer, M, Simmons-O'Brien, E, Vogelsang, G
Formato: case study clinical trial research Journal Article
Publicado: Lippincott Williams & Wilkins 2002 Dec 24
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2002 Dec 24
      vid: 59
      iid: 12
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      pub: Lippincott Williams & Wilkins
      place: Baltimore, Maryland
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        136640040
        10.1212/01.wnl.0000037480.59194.85
        NLM12499476
        136640040
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      tig:
        atl: Thalidomide-induced neuropathy.
      aug:
        au:
          Chaudhry, V
          Cornblath, D R
          Corse, A
          Freimer, M
          Simmons-O'Brien, E
          Vogelsang, G
        affil: Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, MD, USA
      sug:
        subj:
          Neurotoxicity Syndromes Pathology
          Thalidomide Adverse Effects
          Immunosuppressive Agents Adverse Effects
          Thalidomide Therapeutic Use
          Tibial Nerve Pathology
          Middle Age
          Immunosuppressive Agents Therapeutic Use
          Female
          Human
          Neural Conduction Drug Effects
          Neurologic Examination
          Neuropsychological Tests
          Neurotoxicity Syndromes Physiopathology
          Sensation Disorders Physiopathology
          Sensation Disorders Chemically Induced
          Graft Versus Host Disease Prevention and Control
          Bone Marrow Transplantation
          Male
          Paresthesia Chemically Induced
          Adult
          Clinical Trials
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
          Scales
          Middle Aged: 45-64 years
          Adult: 19-44 years
          Female
          Male
      ab: Background: Thalidomide is effective for the treatment of some refractory dermatologic and oncologic diseases. Toxic neuropathy limits its use, as embryopathy can be avoided by contraceptive measures.Objective: To describe the clinical, electrophysiologic, and pathologic features of thalidomide-induced peripheral neuropathy.Methods: Clinical and electrophysiologic examinations were performed in seven patients with thalidomide-induced peripheral neuropathy. Thalidomide was used for graft-vs-host disease, pyoderma gangrenosum, and discoid lupus with dosages ranging from 100 to 1,200 mg/day for 5 to 16 months (cumulative dosages of 24 to 384 g).Results: All seven patients had clinical and electrophysiologic evidence of a sensory more than motor, axonal, length-dependent polyneuropathy that presented as painful paresthesias or numbness. Sural nerve biopsies, done in three patients, showed evidence of Wallerian degeneration and loss of myelinated fibers. The symptoms, signs, and electrophysiologic data correlated with total cumulative dose of thalidomide.Conclusions: Thalidomide induces a dose-dependent sensorimotor length-dependent axonal neuropathy; it should be judiciously used with close neurologic monitoring.
      pubtype: Academic Journal
      doctype:
        case study
        clinical trial
        research
        Journal Article
      ougenre: Article
    language: English
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