A parthenogenetic quasi-program causes teratoma-like tumors during aging in wild-type C. elegans.

A long-standing belief is that aging (senescence) is the result of stochastic damage accumulation. Alternatively, senescent pathology may also result from late-life, wild-type gene action (i.e., antagonistic pleiotropy, as argued by Williams) leading to non-adaptive run-on of developmental programs...

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Publicado en:	NPJ Aging & Mechanisms of Disease Vol. 4; no. 1
Autores principales:	Wang, Hongyuan, Zhao, Yuan, Ezcurra, Marina, Benedetto, Alexandre, Gilliat, Ann F., Hellberg, Josephine, Ren, Ziyu, Galimov, Evgeniy R., Athigapanich, Trin, Girstmair, Johannes, Telford, Maximilian J., Dolphin, Colin T., Zhang, Zhizhou, Gems, David
Formato:	Journal Article
Publicado:	Springer Nature 2018
Acceso en línea:	Ver este registro en EBSCOhost