The Helicobacter pylori Urease Virulence Factor Is Required for the Induction of Hypoxia-Induced Factor-1α in Gastric Cells.

Chronic Helicobacter pylori infection increases the risk of gastric cancer and induction of hypoxia-induced factor (HIF), which is frequently associated with the development and progression of several types of cancer. We recently showed that H. pylori activation of the PI3K-AKT-mTOR pathway in gastr...

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Publicado en:Cancers Vol. 11; no. 6; pp. 799 - 800
Autores principales: Valenzuela-Valderrama, Manuel, Cerda-Opazo, Paulina, Backert, Steffen, González, María Fernanda, Carrasco-Véliz, Nicolás, Jorquera-Cordero, Carla, Wehinger, Sergio, Canales, Jimena, Bravo, Denisse, Quest, Andrew F. G.
Formato: pictorial research tables/charts Journal Article
Publicado: MDPI Jun2019
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jun2019
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      pub: MDPI
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        10.3390/cancers11060799
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        atl: The Helicobacter pylori Urease Virulence Factor Is Required for the Induction of Hypoxia-Induced Factor-1α in Gastric Cells.
      aug:
        au:
          Valenzuela-Valderrama, Manuel
          Cerda-Opazo, Paulina
          Backert, Steffen
          González, María Fernanda
          Carrasco-Véliz, Nicolás
          Jorquera-Cordero, Carla
          Wehinger, Sergio
          Canales, Jimena
          Bravo, Denisse
          Quest, Andrew F. G.
        affil: Laboratorio de Microbiología Celular, Instituto de Innovación e Investigación en Salud, Facultad de Ciencias de la Salud, Universidad Central de Chile, Santiago 8320000, Chile
      sug:
        subj:
          Helicobacter Infections Complications
          Stomach Neoplasms Risk Factors
          Urease Metabolism
          Transcription Factors
          Virulence
          Animal Studies
          Rats
          Mutation
          Staining and Labeling Methods
          Polymers Analysis
          Sodium
          Electrophoresis Methods
          Gene Expression
          Adenocarcinoma
          Antibodies
          Toll-Like Receptors
          Transferases
          Microbial Culture and Sensitivity Tests
      ab: Chronic Helicobacter pylori infection increases the risk of gastric cancer and induction of hypoxia-induced factor (HIF), which is frequently associated with the development and progression of several types of cancer. We recently showed that H. pylori activation of the PI3K-AKT-mTOR pathway in gastric cells increased HIF-1α expression. Here, we identified the H. pylori virulence factor responsible for HIF-1α induction. A mutant of the H. pylori 84-183 strain was identified with reduced ability to induce HIF-1α. Coomassie blue staining of extracts from these bacteria separated by sodium dodecyl sulfate polyacrylamide gel electrophoresis (SDS-PAGE) revealed poor expression of urease subunits that correlated with reduced urease activity. This finding was confirmed in the 26695 strain, where urease mutants were unable to induce HIF-1α expression. Of note, HIF-1α induction was also observed in the presence of the urease inhibitor acetohydroxamic acid at concentrations (of 20 mM) that abrogated urease activity in bacterial culture supernatants, suggesting that enzymatic activity of the urease is not required for HIF-1α induction. Finally, the pre-incubation of the human gastric adenocarcinoma cell line AGS with blocking antibodies against Toll-like receptor-2 (TLR2), but not TLR4, prevented HIF-1α induction. In summary, these results reveal a hitherto unexpected role for the urease protein in HIF-1α induction via TLR2 activation following H. pylori infection of gastric cells.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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