Poststreptococcal acute glomerulonephritis: fact and controversy.

Poststreptococcal acute glomerulonephritis is prototypic of the immunologic glomerulonephritides. It most commonly follows streptococcal infection of the pharynx or skin. The diagnosis is usually not difficult when a nephritic clinical presentation (with such manifestations as hematuria, edema, and...

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Publicado en:Annals of Internal Medicine Vol. 91; no. 1; pp. 76 - 87
Autores principales: Nissenson, A R, Baraff, L J, Fine, R N, Knutson, D W
Formato: research review Journal Article
Publicado: American College of Physicians Jul79
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jul79
      vid: 91
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      pub: American College of Physicians
      place: Philadelphia, Pennsylvania
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        10.7326/0003-4819-91-1-76
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        atl: Poststreptococcal acute glomerulonephritis: fact and controversy.
      aug:
        au:
          Nissenson, A R
          Baraff, L J
          Fine, R N
          Knutson, D W
      sug:
        subj:
          Streptococcal Infections Immunology
          Streptococcal Infections Microbiology
          Glomerulonephritis Etiology
          Pyoderma Diagnosis
          Kidney Failure, Chronic Etiology
          Child, Preschool
          Glomerulonephritis Complications
          Adolescence
          Acute Disease
          Glomerulonephritis Immunology
          Antigens
          Diagnosis, Differential
          Pharyngitis Diagnosis
          Human
          Glomerulonephritis Diagnosis
          Child
          Rheumatic Fever Etiology
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
          Clinical Assessment Tools
          Scales
          Child, Preschool: 2-5 years
          Adolescent: 13-18 years
          Child: 6-12 years
      ab: Poststreptococcal acute glomerulonephritis is prototypic of the immunologic glomerulonephritides. It most commonly follows streptococcal infection of the pharynx or skin. The diagnosis is usually not difficult when a nephritic clinical presentation (with such manifestations as hematuria, edema, and hypertension) is associated with serologic evidence of recent streptococcal infection and a depressed serum complement concentration. Currently, however, the nephritogenic antigen(s) has not been identified and has not been shown to be the same antigen for all nephritogenic streptococci; it may not even be a part of the infecting organism. The development of a vaccine to prevent this illness from occurring is therefore still not possible. Whether poststreptococcal acute glomerulonephritis progresses to chronic renal failure is still uncertain. Painstaking laboratory research together with careful, prospective long-term follow-up studies of patients with poststreptococcal acute glomerulonephritis may provide some of the answers to these critical questions.
      pubtype: Academic Journal
      doctype:
        research
        review
        Journal Article
      ougenre: Article
    language: English
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