Detection of a novel ryanodine receptor subtype 1 mutation (R328W) in a malignant hyperthermia family by sequencing of a leukocyte transcript.

Background: To determine whether malignant hyperthermia (MH) susceptibility in a Canadian pedigree is associated with a mutation in the ryanodine receptor subtype 1 (RYR1) gene, the complete RYR1 transcript obtained from the leukocytes of one MH-susceptible family member was sequenced, using a newly...

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Published in:Anesthesiology Vol. 99; no. 2; pp. 297 - 303
Main Authors: Loke, Julian C P, Kraev, Natasha, Sharma, Parveen, Du, GuoGuang, Patel, Leena, Kraev, Alexander, MacLennan, David H
Format: research Journal Article
Published: Lippincott Williams & Wilkins Aug2003
Online Access:View this record in EBSCOhost
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      dt: Aug2003
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      pub: Lippincott Williams & Wilkins
      place: Baltimore, Maryland
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        atl: Detection of a novel ryanodine receptor subtype 1 mutation (R328W) in a malignant hyperthermia family by sequencing of a leukocyte transcript.
      aug:
        au:
          Loke, Julian C P
          Kraev, Natasha
          Sharma, Parveen
          Du, GuoGuang
          Patel, Leena
          Kraev, Alexander
          MacLennan, David H
        affil: Malignant Hyperthermia Investigation Unit, Banting and Best Department of Medical Research, University of Toronto, Canada
      sug:
        subj:
          Calcium
          Malignant Hyperthermia
          Leukocytes
          Cell Line
          DNA
          Amino Acids
          Pedigree
          Human
          RNA
          Mutation
          Muscle, Skeletal Drug Effects
          Caffeine
          Muscle Contraction Drug Effects
          Reverse Transcriptase Polymerase Chain Reaction
          Anesthetics, Inhalation
          Phosphodiesterase Inhibitors
          Halothane
          Validation Studies
          Comparative Studies
          Evaluation Research
          Multicenter Studies
      ab: Background: To determine whether malignant hyperthermia (MH) susceptibility in a Canadian pedigree is associated with a mutation in the ryanodine receptor subtype 1 (RYR1) gene, the complete RYR1 transcript obtained from the leukocytes of one MH-susceptible family member was sequenced, using a newly developed protocol.Methods: RNA was extracted from leukocytes and converted into complementary DNA. Overlapping fragments of RYR1 complementary DNA were amplified by the polymerase chain reaction and used for double-strand sequencing to find a single mutation likely to be causal of MH susceptibility. Inheritance of the mutation in the family was studied by restriction endonuclease analysis and/or sequencing of genomic DNA and compared to available caffeine halothane contracture test data. The mutation was introduced into rabbit RYR1 complementary DNA, the complementary DNA was expressed in human embryonic kidney line 293 cells, and Ca2+ release by the mutant Ca2+ release channel was measured following the addition of caffeine and halothane.Results: A novel arginine 328 to tryptophan mutation in RYR1 was detected by direct sequencing of the RYR1 transcript from leukocytes of one MH-susceptible individual. A causal role for this mutation in MH is indicated by cosegregation of the mutation with the MH-susceptible phenotype within the family and by the demonstration that the mutant channel has increased sensitivity to both caffeine and halothane.Conclusions: The feasibility of using complete RYR1 transcripts from leukocytes for sequence analysis offers an efficient and noninvasive method for scanning RYR1 for novel mutations.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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