Intestinal Microbial Products From Alcohol‐Fed Mice Contribute to Intestinal Permeability and Peripheral Immune Activation.

Background: Alcohol use causes significant disruption of intestinal microbial communities, yet exactly how these dysbiotic communities interact with the host is unclear. We sought to understand the role of microbial products associated with alcohol dysbiosis in mice on intestinal permeability and im...

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Publicado en:Alcoholism: Clinical & Experimental Research Vol. 43; no. 10; pp. 2122 - 2134
Autores principales: Samuelson, Derrick R., Gu, Min, Shellito, Judd E., Molina, Patricia E., Taylor, Christopher M., Luo, Meng, Welsh, David A.
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell Oct2019
Acceso en línea:Ver este registro en EBSCOhost
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      jtl: Alcoholism: Clinical & Experimental Research
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      dt: Oct2019
      vid: 43
      iid: 10
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Intestinal Microbial Products From Alcohol‐Fed Mice Contribute to Intestinal Permeability and Peripheral Immune Activation.
      aug:
        au:
          Samuelson, Derrick R.
          Gu, Min
          Shellito, Judd E.
          Molina, Patricia E.
          Taylor, Christopher M.
          Luo, Meng
          Welsh, David A.
        affil: Section of Pulmonary/Critical Care and Allergy/Immunology, Department of Internal Medicine, Louisiana State University Health Sciences Center, New Orleans Louisiana
      sug:
        subj:
          Binge Drinking Metabolism
          Binge Drinking Microbiology
          Binge Drinking Immunology
          Intestines Metabolism
          Gut Microbiota Metabolism
          Intestines Immunology
          Gut Microbiota Immunology
          Animal Studies
          Mice
          In Vitro Studies
          Models, Biological
          Binge Drinking
          Epithelial Cells
          Electric Stimulation Methods
          Flow Cytometry
          Blood Cells Analysis
          Antigens, Surface Analysis
          Descriptive Statistics
      ab: Background: Alcohol use causes significant disruption of intestinal microbial communities, yet exactly how these dysbiotic communities interact with the host is unclear. We sought to understand the role of microbial products associated with alcohol dysbiosis in mice on intestinal permeability and immune activation in an in vitro model system. Methods: Microbiota samples from binge‐on‐chronic alcohol‐fed and pair‐fed male and female mice were cultured in Gifu Anaerobic Broth for 24 hours under anaerobic conditions. Live/whole organisms were removed, and microbial products were collected and added to human peripheral blood mononuclear cells (PBMCs) or polarized C2BBe1 intestinal epithelial monolayers. Following stimulation, transepithelial electrical resistance (TEER) was measured using a volt/ohm meter and immune activation of PBMC was assessed via flow cytometry. Results: Microbial products from male and female alcohol‐fed mice significantly decreased TEER (mean percentage change from baseline alcohol‐fed 0.86 Ω/cm2 vs. pair‐fed 1.10 Ω/cm2) compared to microbial products from control mice. Following ex vivo stimulation, immune activation of PBMC was assessed via flow cytometry. We found that microbial products from alcohol‐fed mice significantly increased the percentage of CD38+ CD4+ (mean alcohol‐fed 17.32% ± 0.683% standard deviation (SD) vs. mean pair‐fed 14.2% ± 1.21% SD, p < 0.05) and CD8+ (mean alcohol‐fed 20.28% ± 0.88% SD vs. mean pair‐fed 12.58% ± 3.59% SD, p < 0.05) T cells. Conclusions: Collectively, these data suggest that microbial products contribute to immune activation and intestinal permeability associated with alcohol dysbiosis. Further, utilization of these ex vivo microbial product assays will allow us to rapidly assess the impact of microbial products on intestinal permeability and immune activation and to identify probiotic therapies to ameliorate these defects.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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