Vedolizumab-Induced De Novo Extraintestinal Manifestations.

Background: Vedolizumab is an α4β7 integrin antagonist with gut-specific effects on lymphocyte and monocyte trafficking. Although the treatment is beneficial for inflammatory bowel disease (IBD), the effects of vedolizumab on extraintestinal manifestations (EIMs) have not been well described. The gu...

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Publicado en:Gastroenterology & Hepatology Vol. 16; no. 2; pp. 74 - 81
Autores principales: Diaz, Liege I., Keihanian, Tara, Schwartz, Ingrid, Su Bin Kim, Calmet, Fernando, Quintero, Maria Alejandra, Abreu, Maria T.
Formato: research tables/charts Journal Article
Publicado: Gastro-Hep Communications, Inc Feb2020
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Feb2020
      vid: 16
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      pub: Gastro-Hep Communications, Inc
      place: New York, New York
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        atl: Vedolizumab-Induced De Novo Extraintestinal Manifestations.
      aug:
        au:
          Diaz, Liege I.
          Keihanian, Tara
          Schwartz, Ingrid
          Su Bin Kim
          Calmet, Fernando
          Quintero, Maria Alejandra
          Abreu, Maria T.
        affil: Advanced gastroenterology fellow in the Department of Gastrointestinal Oncology at the Moffitt Cancer Center in Tampa, Florida
      sug:
        subj:
          Antibodies, Monoclonal Adverse Effects
          Arthralgia Risk Factors
          Fistula Risk Factors
          Pyoderma Gangrenosum Risk Factors
          Inflammatory Bowel Diseases Drug Therapy
          Human
          Retrospective Design
          Record Review
          Colonoscopy
          Crohn Disease Risk Factors
          Colitis, Ulcerative Risk Factors
          Inflammation
          Lymphocytes Drug Effects
          Monocytes Drug Effects
          Descriptive Statistics
      ab: Background: Vedolizumab is an α4β7 integrin antagonist with gut-specific effects on lymphocyte and monocyte trafficking. Although the treatment is beneficial for inflammatory bowel disease (IBD), the effects of vedolizumab on extraintestinal manifestations (EIMs) have not been well described. The gut-specific effects of the medication may have diverse outcomes on EIMs. We hypothesize that EIMs may be unmasked by systemic availability of gut-homing effector cells. Aim: The goal of this study is to describe de novo EIMs of IBD patients who were started on vedolizumab. Methods: A retrospective chart review of 71 patients from January 2011 to October 2017, including clinical and medication history and colonoscopy results, was performed. Results: EIMs occurred in 26.7% of patients who were started on vedolizumab. The most common EIMs were arthralgias, perianal fistula, and pyoderma gangrenosum. There was a trend toward a greater occurrence of EIMs in patients with Crohn's disease compared to ulcerative colitis. Conclusion: Our retrospective study suggests that inhibition of gut-specific effector cells results in activated lymphocytes and/or monocytes that cause inflammation in other tissues. More studies are needed to confirm these observations and to develop biomarkers that predict patients at risk for EIMs and perianal fistulas while on vedolizumab.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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