Precancerous Gastric Lesions with Helicobacter pylori vacA+/babA2+/oipA+ Genotype Increase the Risk of Gastric Cancer.

Objective. The clinical outcomes of gastric diseases such as chronic gastritis, peptic ulcer, and gastric cancer have been attributed to the interplay of virulence factors of Helicobacter pylori (H. pylori), host genetic susceptibility, and host immune responses. This study investigated the presence...

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Publicado en:BioMed Research International pp. 1 - 9
Autores principales: Bartpho, Theeraya Simawaranon, Wattanawongdon, Wareeporn, Tongtawee, Taweesak, Paoin, Chatchanok, Kangwantas, Kokiet, Dechsukhum, Chavaboon
Formato: research tables/charts Journal Article
Publicado: Wiley-Blackwell 2/19/2020
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2/19/2020
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2020/7243029
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        atl: Precancerous Gastric Lesions with Helicobacter pylori vacA+/babA2+/oipA+ Genotype Increase the Risk of Gastric Cancer.
      aug:
        au:
          Bartpho, Theeraya Simawaranon
          Wattanawongdon, Wareeporn
          Tongtawee, Taweesak
          Paoin, Chatchanok
          Kangwantas, Kokiet
          Dechsukhum, Chavaboon
        affil: Translational Medicine Programs, Institute of Medicine, Suranaree University of Technology, Nakhon Ratchasima 30000, Thailand
      sug:
        subj:
          Helicobacter Infections Complications
          Virulence Evaluation
          Genotype Evaluation
          Precancerous Conditions Risk Factors
          Stomach Neoplasms Risk Factors
          Stomach Neoplasms Familial and Genetic
          Precancerous Conditions Familial and Genetic
          Risk Assessment
          Human
          Chronic Disease
          Gastritis Diagnosis
          Gastritis, Atrophic Diagnosis
          Intestines Pathology
          Metaplasia Diagnosis
          Gastritis Surgery
          Gastritis, Atrophic Surgery
          Metaplasia Surgery
          Endoscopy Methods
          Surgery, Operative Methods
          Polymerase Chain Reaction Methods
          Multiple Regression
          Stomach Neoplasms Prognosis
          Survival
          Prevalence
          Odds Ratio
          Confidence Intervals
          Gastritis Risk Factors
      ab: Objective. The clinical outcomes of gastric diseases such as chronic gastritis, peptic ulcer, and gastric cancer have been attributed to the interplay of virulence factors of Helicobacter pylori (H. pylori), host genetic susceptibility, and host immune responses. This study investigated the presence of cagA, vacA, iceA2, babA2, and oipA genes and their association with clinical outcomes. Methods. Chronic gastritis, atrophic gastritis, and intestinal metaplasia specimens were obtained from patients who underwent endoscopy and surgical resection between January 2017 and December 2018; specimens from gastric cancer patients treated between January 2014 and December 2018 were also added. H. pylori infection and virulence genes (cagA, vacA, iceA2, babA2, and oipA) were determined using real-time PCR. The association between H. pylori genotypes and clinical outcomes were evaluated using multivariate regression model analysis. The overall survival of gastric cancer patients was compared between genotype combinations. Results. H. pylori was positive in 166 patients with chronic gastritis, precancerous gastric lesions, and gastric cancer. The genes vacA, babA2, and oipA were most prevalent in chronic gastritis (73%), precancerous gastric lesions (62%), and gastric cancer (91%), respectively. The vacA, babA2, and oipA genes were associated with increased risk of gastric cancer (OR = 1.23; 95% CI = 1.13–3.32; P = 0.033 , OR = 2.64; 95% CI = 1.44–4.82, P = 0.024 , and OR = 2.79; 95% CI = 1.58–5.41; P = 0.031 , respectively). Interestingly, H. pylori vacA+/babA2+/oipA+ genotype infection was associated with increased risk of gastric cancer (OR = 3.85, 95% CI = 1.67–5.77, P = 0.014). Conclusion. In this present study, we reported on the virulence genes of H. pylori infection to reveal their association with increased risk of chronic gastritis, precancerous gastric lesions, and gastric cancer. Precancerous gastric lesions with H. pylori vacA+/babA2+/oipA+ genotype increased the risk of gastric cancer.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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