Alcohol use disorders and the brain.

A diagnosis of alcohol use disorder is associated with a higher risk of dementia, but a dose–response relationship between alcohol intake consumption and cognitive impairment remains unclear. Alcohol is associated with a range of effects on the central nervous system at different doses and acts on a...

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Publicado en:Addiction Vol. 115; no. 8; pp. 1580 - 1590
Autores principales: Rao, Rahul, Topiwala, Anya
Formato: review tables/charts Journal Article
Publicado: Wiley-Blackwell Aug2020
Acceso en línea:Ver este registro en EBSCOhost
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Alcohol use disorders and the brain.
      aug:
        au:
          Rao, Rahul
          Topiwala, Anya
        affil: Institute of Psychiatry, Psychology and Neuroscience, Department of Old Age Psychiatry, London, UK
      sug:
        subj:
          Brain Physiology
          Central Nervous System Physiology
          Alcohol-Related Disorders Complications
          Dementia Risk Factors
          Dose-Response Relationship
          Cognition Disorders Risk Factors
          Wernicke's Encephalopathy Risk Factors
          Brain Injuries Risk Factors
          Seizures Risk Factors
          Stroke Risk Factors
          Hepatic Encephalopathy Risk Factors
          Korsakoff Syndrome Risk Factors
          Alcohol-Related Disorders Epidemiology
          Alcohol-Related Disorders Therapy
          Quality of Life
          Behavior
      ab: A diagnosis of alcohol use disorder is associated with a higher risk of dementia, but a dose–response relationship between alcohol intake consumption and cognitive impairment remains unclear. Alcohol is associated with a range of effects on the central nervous system at different doses and acts on a number of receptors. Acute disorders include Wernicke's encephalopathy (WE), traumatic brain injury, blackouts, seizures, stroke and hepatic encephalopathy. The most common manifestations of chronic alcohol consumption are Korsakoff's syndrome (KS) and alcohol‐related dementia (ARD). There is limited evidence for benefit from memantine in the treatment of ARD, but stronger evidence for the use of high‐dose parenteral thiamine in the progression of neuropsychiatric symptoms for WE. Accumulating evidence exists for pharmacological treatment in the prevention of hepatic encephalopathy. Rehabilitation of people with ARD may take several years, and requires an approach that addresses physical and psychosocial factors.
      pubtype: Academic Journal
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      ougenre: Article
    language: English
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