Influence of CYP2D6*5 and *10 polymorphism on the pharmacokinetics of nebivolol in healthy Chinese subjects.

What is known and objective: Nebivolol, a selective β1 adrenoreceptor antagonist, is predominantly metabolized by cytochrome P450 (CYP)2D6 and shows a wide interindividual variability in pharmacokinetics. The present study was conducted to evaluate the effects of the major CYP2D6 polymorphisms on ne...

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Publicado en:Journal of Clinical Pharmacy & Therapeutics Vol. 45; no. 4; pp. 632 - 638
Autores principales: Guo, Lifang, Wang, Shumin, Wan, Zirui, Ni, Siyang, Xu, Benshan, Zhao, Xiuli, Liu, Lihong
Formato: research tables/charts Journal Article
Publicado: Wiley-Blackwell Aug2020
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Aug2020
      vid: 45
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1111/jcpt.13155
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        atl: Influence of CYP2D6*5 and *10 polymorphism on the pharmacokinetics of nebivolol in healthy Chinese subjects.
      aug:
        au:
          Guo, Lifang
          Wang, Shumin
          Wan, Zirui
          Ni, Siyang
          Xu, Benshan
          Zhao, Xiuli
          Liu, Lihong
        affil: Pharmacy Department, Beijing Chao‐Yang Hospital, Capital Medical University, Beijing, China
      sug:
        subj:
          Nebivolol Hydrochloride Pharmacokinetics
          Polymorphism, Genetic
          Chinese Persons
          Human
          Genotype
          Chromatography, High Pressure Liquid
          Mass Spectrometry
          Unpaired T-Tests
          Analysis of Variance
          Nebivolol Hydrochloride Blood
          Polymorphism, Single Nucleotide
      ab: What is known and objective: Nebivolol, a selective β1 adrenoreceptor antagonist, is predominantly metabolized by cytochrome P450 (CYP)2D6 and shows a wide interindividual variability in pharmacokinetics. The present study was conducted to evaluate the effects of the major CYP2D6 polymorphisms on nebivolol disposition in healthy Chinese volunteers. Methods: Twenty‐eight volunteers were enrolled and classified as CYP2D6*1/*1, CYP2D6*1/*10, CYP2D6*10/*10 and CYP2D6*5 carriers according to their genotypes. The concentration of nebivolol was determined by high‐performance liquid chromatography‐tandem mass spectrometry. The association between the pharmacokinetic parameters and genotypes was evaluated using the unpaired t test or analysis of variance. Results and discussion: We evaluated the effects of CYP2D6*5 and *10 polymorphism on the pharmacokinetics of nebivolol. Plasma nebivolol peak concentration and area under the curve (AUC(0‐48 h) and AUC(0‐∞)) were significantly higher in subjects with CYP2D6*5 and CYP2D6*10/*10 polymorphism than those in subjects with wild‐type CYP2D6 (CYP2D6*1/*1), whereas its plasma clearance was significantly lower in the CYP2D6*10/*10 and CYP2D6*5 carriers. No significant differences in the peak time and terminal half‐life of nebivolol were observed among CYP2D6*10/*10, CYP2D6*1/*1 and CYP2D6*5 carriers. What is new and conclusion: Both CYP2D6*5 and *10 polymorphism altered the pharmacokinetics of nebivolol in healthy Chinese volunteers. Further studies are required to investigate the effects of these single‐nucleotide polymorphisms on the pharmacokinetics, pharmacodynamics and toxicity of nebivolol.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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