Detection of Pathogenic Isoforms of IKZF1 in Leukemic Cell Lines and Acute Lymphoblastic Leukemia Samples: Identification of a Novel Truncated IKZF1 Transcript in SUP-B15.

Simple Summary: Abnormal RNA splicing plays a fundamental role in leukemogenesis in acute lymphoblastic leukemia (ALL). Many cases of high-risk B-cell ALL cases, including BCR-ABL1+ and BCR-ABL1-like ALL, share a common molecular mechanism of aberrant fusion transcripts involving tyrosine kinase gen...

Descripción completa

Detalles Bibliográficos
Publicado en:Cancers Vol. 12; no. 11; pp. 3161 - 3162
Autores principales: Zhao, Weiqiang, Li, Ying, Yao, Chenjiao, Zhang, Guojuan, Zhao, Kevin Y., Chen, Wei, Ru, Peng, Pan, Xiaokang, Tu, Huolin, Jones, Daniel
Formato: pictorial research tables/charts Journal Article
Publicado: MDPI Nov2020
Acceso en línea:Ver este registro en EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=147285172&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 147285172
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        20726694
        B74B
      jtl: Cancers
      issn: 20726694
      maglogo: N
    pubinfo:
      dt: Nov2020
      vid: 12
      iid: 11
      pid: 97109
      pub: MDPI
    artinfo:
      ui:
        147285172
        147285172
        147285172
        10.3390/cancers12113161
        147285172
      ppf: 3161
      ppct: 1
      formats:
      tig:
        atl: Detection of Pathogenic Isoforms of IKZF1 in Leukemic Cell Lines and Acute Lymphoblastic Leukemia Samples: Identification of a Novel Truncated IKZF1 Transcript in SUP-B15.
      aug:
        au:
          Zhao, Weiqiang
          Li, Ying
          Yao, Chenjiao
          Zhang, Guojuan
          Zhao, Kevin Y.
          Chen, Wei
          Ru, Peng
          Pan, Xiaokang
          Tu, Huolin
          Jones, Daniel
        affil: The James Comprehensive Cancer Center and Solove Research Institute, The Ohio State University, Columbus, OH 43210, USA
      sug:
        subj:
          Leukemia, Lymphocytic, Acute Physiopathology
          Leukemia, Lymphocytic, Acute Prognosis
          Cell Line, Tumor Analysis
          Neoplastic Processes
          RNA
          Mutation
          Transcription Factors
          Human
          Adult
          Oncogenes
          Protein-Tyrosine Kinases
          Genetic Techniques
          Interleukins
          Gene Expression Profiling
          Histone Deacetylases Antagonists and Inhibitors
          Age Factors
          Chromosome Aberrations
          Adult: 19-44 years
      ab: Simple Summary: Abnormal RNA splicing plays a fundamental role in leukemogenesis in acute lymphoblastic leukemia (ALL). Many cases of high-risk B-cell ALL cases, including BCR-ABL1+ and BCR-ABL1-like ALL, share a common molecular mechanism of aberrant fusion transcripts involving tyrosine kinase genes combined with dysregulation of the transcription factor and lymphocyte differentiation factor IKZF1. Dysfunction of IKZF1 in ALL is caused by mutation and gene deletion but also alternative splicing resulting in exon skipping with production of aberrant IKZF1 proteins. We report here an assay to detect aberrantly spliced isoforms of IKZF1 in ALL to assist in diagnosis, outcome prediction, and therapy selection in ALL and the identification of a novel altered IKZF1 product in a model ALL cell line. Leukemia-associated alternative splicing of IKZF1 can result in proteins with loss of one to four copies of its N-terminal zinc finger domains (N-ZnF). The best characterized pathogenic splice isoforms, Ik-6 and Ik-8, have been commonly found in BCR-ABL1+ acute lymphoblastic leukemia (ALL) and a subset of BCR-ABL1-like ALL. Infantile and childhood ALL that express these pathogenic IKZF1 isoforms have shown inferior clinical outcomes and can be resistant to tyrosine kinase inhibitors. Using ALL cell lines, we designed and validated a method to detect abnormal IKZF1 transcripts. In the SUP-B15 leukemia cell line, we noted novel IKZF1 transcripts that include both an Ik-6 splice and a transcript with a 14 base pair insertion at the C-terminus. There was also increased IKZF2 protein in SUP-B15 as compared to other ALL lines. Expression of Ik-6 could be suppressed by treatment with the pro-apoptotic type II histone deacetylase inhibitor givinostat. In 17 adult ALL samples, we noted the Ik-6 isoforms in 6 of 15 BCR-ABL1−, and 1 of 2 BCR-ABL1+ cases, with Ik-8 also expressed in one case. Cases with Ik-6 expression showed inferior survival as well as older age at presentation, lower expression of CD10 and more commonly a diploid karyotype.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N