Differential transcript usage unravels gene expression alterations in Alzheimer's disease human brains.

Alzheimer's disease (AD) is the leading cause of dementia in aging individuals. Yet, the pathophysiological processes involved in AD onset and progression are still poorly understood. Among numerous strategies, a comprehensive overview of gene expression alterations in the diseased brain could contr...

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Publicado en:NPJ Aging & Mechanisms of Disease Vol. 7; no. 1; pp. 1 - 16
Autores principales: Marques-Coelho, Diego, Iohan, Lukas da Cruz Carvalho, Melo de Farias, Ana Raquel, Flaig, Amandine, Letournel, Franck, Martin-Négrier, Marie-Laure, Chapon, Françoise, Faisant, Maxime, Godfraind, Catherine, Maurage, Claude-Alain, Deramecourt, Vincent, Duchesne, Mathilde, Meyronnet, David, Streichenberger, Nathalie, de Paula, André Mauès, Rigau, Valérie, Vandenbos-Burel, Fanny, Duyckaerts, Charles, Seilhean, Danielle, Milin, Serge
Formato: Journal Article
Publicado: Springer Nature 1/4/2021
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 1/4/2021
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      pub: Springer Nature
      place: New York, New York
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        10.1038/s41514-020-00052-5
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        atl: Differential transcript usage unravels gene expression alterations in Alzheimer's disease human brains.
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          Marques-Coelho, Diego
          Iohan, Lukas da Cruz Carvalho
          Melo de Farias, Ana Raquel
          Flaig, Amandine
          Letournel, Franck
          Martin-Négrier, Marie-Laure
          Chapon, Françoise
          Faisant, Maxime
          Godfraind, Catherine
          Maurage, Claude-Alain
          Deramecourt, Vincent
          Duchesne, Mathilde
          Meyronnet, David
          Streichenberger, Nathalie
          de Paula, André Mauès
          Rigau, Valérie
          Vandenbos-Burel, Fanny
          Duyckaerts, Charles
          Seilhean, Danielle
          Milin, Serge
        affil: Brain Institute, Federal University of Rio Grande do Norte, Av. Nascimento de Castro, 2155, Natal, Brazil
      sug:
      ab: Alzheimer's disease (AD) is the leading cause of dementia in aging individuals. Yet, the pathophysiological processes involved in AD onset and progression are still poorly understood. Among numerous strategies, a comprehensive overview of gene expression alterations in the diseased brain could contribute for a better understanding of the AD pathology. In this work, we probed the differential expression of genes in different brain regions of healthy and AD adult subjects using data from three large transcriptomic studies: Mayo Clinic, Mount Sinai Brain Bank (MSBB), and ROSMAP. Using a combination of differential expression of gene and isoform switch analyses, we provide a detailed landscape of gene expression alterations in the temporal and frontal lobes, harboring brain areas affected at early and late stages of the AD pathology, respectively. Next, we took advantage of an indirect approach to assign the complex gene expression changes revealed in bulk RNAseq to individual cell types/subtypes of the adult brain. This strategy allowed us to identify previously overlooked gene expression changes in the brain of AD patients. Among these alterations, we show isoform switches in the AD causal gene amyloid-beta precursor protein (APP) and the risk gene bridging integrator 1 (BIN1), which could have important functional consequences in neuronal cells. Altogether, our work proposes a novel integrative strategy to analyze RNAseq data in AD and other neurodegenerative diseases based on both gene/transcript expression and regional/cell-type specificities.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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