Absence of significant association between UGT2B4 genetic variants and the susceptibility to anti‐tuberculosis drug‐induced liver injury in a Western Chinese population.

What is known and objective: Combination regimens of six‐month duration may increase the incidence of anti‐tuberculosis drug‐induced liver injury (ATLI), which is clinically characterized by mild cholestasis and hepatocanalicular lesions. UGT2B4 is a predominant UDP‐glucuronosyltransferase enzyme in...

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Publicado en:Journal of Clinical Pharmacy & Therapeutics Vol. 46; no. 1; pp. 66 - 74
Autores principales: Chen, Hao, Jiao, Lin, Zhou, Juan, Bai, Hao, Lyu, Mengyuan, Wu, Tao, Wu, Lijuan, Song, Jiajia, Liu, Tangyuheng, Yan, Hong, Ying, Binwu
Formato: research tables/charts Journal Article
Publicado: Wiley-Blackwell Feb2021
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Feb2021
      vid: 46
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        147968586
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        10.1111/jcpt.13132
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        atl: Absence of significant association between UGT2B4 genetic variants and the susceptibility to anti‐tuberculosis drug‐induced liver injury in a Western Chinese population.
      aug:
        au:
          Chen, Hao
          Jiao, Lin
          Zhou, Juan
          Bai, Hao
          Lyu, Mengyuan
          Wu, Tao
          Wu, Lijuan
          Song, Jiajia
          Liu, Tangyuheng
          Yan, Hong
          Ying, Binwu
        affil: Department of Laboratory Medicine, West China Hospital, Sichuan University, Chengdu, China
      sug:
        subj:
          Polymorphism, Genetic
          Disease Susceptibility
          Antitubercular Agents Adverse Effects
          Liver Diseases Chemically Induced
          Liver Diseases Risk Factors
          Hepatotoxicity Risk Factors
          Risk Assessment
          Human
          China
          Genomics
          DNA
          Blood
          Genetic Profile
          Hospitals China
          Phenotype
          Alleles
          Cholestasis Chemically Induced
          Polymorphism, Single Nucleotide
      ab: What is known and objective: Combination regimens of six‐month duration may increase the incidence of anti‐tuberculosis drug‐induced liver injury (ATLI), which is clinically characterized by mild cholestasis and hepatocanalicular lesions. UGT2B4 is a predominant UDP‐glucuronosyltransferase enzyme in the human liver that plays an important role in the detoxification of bile acids, which yields water‐soluble inactive compounds that can easily be excreted in the bile or urine. This study aimed to investigate the potential association between UGT2B4 variants and the susceptibility to ATLI. Methods: Genomic DNA was extracted from whole blood sample of each patient, and all SNPs were genotyped using an improved multiplex ligation detection reaction method. Clinical symptoms and laboratory results were recorded regularly. Five genetic variants at UGT2B4(rs1131878, rs1966151, rs28361541, rs4557343 and rs79407331) were identified in a prospective study of 118 ATLI cases and 628 non‐ATLI controls. All participants were treated by first‐line anti‐TB drugs in Western China Hospital. The potential association between SNPs, ATLI risk and clinical phenotypes were determined based on the distribution of allelic frequencies and different genetic models. Results and discussion: Statistical comparisons of cases and controls after correction for multiple testing did not yield any significant association between genetic variants at UGT2B4 and risk of ATLI via the analyses of single locus and subgroup differences. What is new and conclusion: This is the first study aimed to investigate the association of UGT2B4 polymorphisms with ATLI risk. Our results revealed that UGT2B4 genetic variants are unlikely to confer susceptibility to ATLI in the Western Chinese Han population.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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