Intestinal function is impaired in patients with Chronic Obstructive Pulmonary Disease.

Gastrointestinal symptoms are prevalent extrapulmonary systemic manifestations of Chronic Obstructive Pulmonary Disease (COPD), but have been rarely studied. We dissected the perturbations in intestinal function in human patients with COPD using comprehensive metabolic and physiological approaches....

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Publicado en:Clinical Nutrition Vol. 40; no. 4; pp. 2270 - 2278
Autores principales: Kirschner, Sarah K., Deutz, Nicolaas E.P., Jonker, Renate, Olde Damink, Steven W.M., Harrykissoon, Rajesh I., Zachria, Anthony J., Dasarathy, Srinivasan, Engelen, Mariëlle P.K.J.
Formato: research Journal Article
Publicado: Elsevier B.V. Apr2021
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Apr2021
      vid: 40
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      pub: Elsevier B.V.
      place: New York, New York
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        10.1016/j.clnu.2020.10.010
        149781231
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        atl: Intestinal function is impaired in patients with Chronic Obstructive Pulmonary Disease.
      aug:
        au:
          Kirschner, Sarah K.
          Deutz, Nicolaas E.P.
          Jonker, Renate
          Olde Damink, Steven W.M.
          Harrykissoon, Rajesh I.
          Zachria, Anthony J.
          Dasarathy, Srinivasan
          Engelen, Mariëlle P.K.J.
        affil: Center for Translational Research in Aging & Longevity, Department of Health & Kinesiology, Texas A&M University, College Station, TX, USA
      sug:
        subj:
          Pulmonary Disease, Chronic Obstructive
          Intestines Physiopathology
          Gut Microbiota Metabolism
          Human
          Nonexperimental Studies
          Confidence Intervals
          Descriptive Statistics
      ab: Gastrointestinal symptoms are prevalent extrapulmonary systemic manifestations of Chronic Obstructive Pulmonary Disease (COPD), but have been rarely studied. We dissected the perturbations in intestinal function in human patients with COPD using comprehensive metabolic and physiological approaches. In this observational study, small intestinal membrane integrity and active carrier-mediated glucose transport were quantified by sugar permeability test in 21 clinically stable patients with moderate to severe COPD (mean FEV 1 , 41.2 (3.2) % of predicted) and 16 healthy control subjects. Protein digestion and absorption was analyzed using stable tracer kinetic methods. Plasma acetate, propionate, and butyrate concentrations were measured as markers of intestinal microbial metabolism. Compared with healthy controls, non carrier-mediated permeability was higher (0.062 (95% CI [0.046, 0.078]) vs. 0.037 (95% CI [0.029, 0.045]), P = 0.009) and active glucose transport lower in COPD (31.4 (95% CI [23.4, 39.4])% vs. 48.0 (95% CI [37.8, 58.3])%, P = 0.010). Protein digestion and absorption was lower in COPD (0.647 (95% CI [0.588, 0.705]) vs. 0.823 (95% CI [0.737, 0.909]), P = 0007), and impairment greater in patients with dyspnea (P = 0.038), exacerbations in preceding year (P = 0.052), and reduced transcutaneous oxygen saturation (P = 0.051), and was associated with reduced physical activity score (P = 0.016) and lower quality of life (P = 0.0007). Plasma acetate concentration was reduced in COPD (41.54 (95% CI [35.17, 47.91]) vs. 80.44 (95% CI [54.59, 106.30]) μmol/L, P = 0.001) suggesting perturbed intestinal microbial metabolism. We conclude that intestinal dysfunction is present in COPD, worsens with increasing disease severity, and is associated with reduced quality of life.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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