MCU That Is Transcriptionally Regulated by Nrf2 Augments Malignant Biological Behaviors in Oral Squamous Cell Carcinoma Cells.

Objective. To clarify the role and molecular mechanism of mitochondrial calcium uniporter (MCU) in the malignant biological behaviors of oral squamous cell carcinoma (OSCC) cells through clinical and cellular experiments. Methods. Immunohistochemistry and qRT-PCR techniques were used to observe the...

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Publicado en:BioMed Research International pp. 1 - 18
Autores principales: Wu, Ran, Zuo, Weiwen, Xu, Xiaoliang, Bi, Lei, Zhang, Chunguang, Chen, Hui, Liu, Hui
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 6/4/2021
Acceso en línea:Ver este registro en EBSCOhost
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      jtl: BioMed Research International
      issn: 23146133
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      dt: 6/4/2021
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        150693063
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        10.1155/2021/6650791
        150693063
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        atl: MCU That Is Transcriptionally Regulated by Nrf2 Augments Malignant Biological Behaviors in Oral Squamous Cell Carcinoma Cells.
      aug:
        au:
          Wu, Ran
          Zuo, Weiwen
          Xu, Xiaoliang
          Bi, Lei
          Zhang, Chunguang
          Chen, Hui
          Liu, Hui
        affil: Department of Stomatology, North China University of Science and Technology Affiliated Hospital, Tangshan, 063000 Hebei, China
      sug:
        subj:
          Carcinoma, Squamous Cell
          Mouth Neoplasms
          Transcription Factors Analysis
          Calcium Analysis
          Mitochondria
          NF-E2-Related Factor 2 Physiology
          Human
          Immunohistochemistry
          Reverse Transcriptase Polymerase Chain Reaction
          Membrane Potentials
          Blotting, Western
          Fluorescent Antibody Technique
      ab: Objective. To clarify the role and molecular mechanism of mitochondrial calcium uniporter (MCU) in the malignant biological behaviors of oral squamous cell carcinoma (OSCC) cells through clinical and cellular experiments. Methods. Immunohistochemistry and qRT-PCR techniques were used to observe the expression of MCU, nuclear factor erythroid 2-related factor 2 (Nrf2), mitochondrial calcium uptake 1 (MICU1), and MICU2 in OSCC and normal tissues. After treatment with si-MCU, spermine, and/or sh-Nrf2, malignant biological behaviors of OSCC cells including proliferation, migration, and apoptosis were detected by clone formation, migration, and mitochondrial membrane potential (MMP) assays. Furthermore, MCU, MICU1, MICU2, Nrf2, and other proteins related to malignant biological behaviors were examined using western blot, immunohistochemistry, and immunofluorescence assays. Results. MCU, Nrf2, and MICU1 were strongly expressed in OSCC as compared to normal tissues, while MICU2 was relatively weakly expressed in OSCC tissues. Knockdown of MCU distinctly weakened proliferation and migration and lowered MMP level in CAL 27 cells. Conversely, its activation reinforced migrated capacity and increased MMP level in CAL 27 cells, which was reversed after cotransfection with sh-Nrf2. After treatment with si-MCU or spermine, Nrf2 expression was not affected in CAL 27 cells. However, MCU expression was distinctly suppressed in CAL 27 cells transfected with sh-Nrf2. Furthermore, knockdown of Nrf2 significantly reversed the increase in expression of MICU1 and MICU2 induced by MCU activation in CAL 27 cells. Conclusion. MCU, as a novel oncogene of OSCC, augments malignant biological behaviors of OSCC cells, which could be transcriptionally regulated by Nrf2.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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