The Level of Mesenchymal-Epithelial Transition Autophosphorylation is Correlated with Esophageal Squamous Cell Carcinoma Migration.

Background: The MET receptor is a critical member of cancer-associated receptor tyrosine kinases and plays an important role in different biological activities, including differentiation, migration, and cell proliferation.Methods: In this study, novel MET inhibitors were introduced and applied on es...

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Publicado en:Iranian Biomedical Journal Vol. 25; no. 4; pp. 243 - 255
Autores principales: Taghehchian, Negin, Moghbeli, Meysam, Mashkani, Baratali, Abbaszadegan, Mohammad Reza
Formato: research Journal Article
Publicado: Pasteur Institute of Iran Jul2021
Acceso en línea:Ver este registro en EBSCOhost
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      jtl: Iranian Biomedical Journal
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      dt: Jul2021
      vid: 25
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      pub: Pasteur Institute of Iran
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        10.52547/ibj.25.4.243
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        151297185
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      tig:
        atl: The Level of Mesenchymal-Epithelial Transition Autophosphorylation is Correlated with Esophageal Squamous Cell Carcinoma Migration.
      aug:
        au:
          Taghehchian, Negin
          Moghbeli, Meysam
          Mashkani, Baratali
          Abbaszadegan, Mohammad Reza
        affil: Department of Chemistry, Faculty of Science, Ferdowsi University of Mashhad, Mashhad, Iran
      sug:
        subj:
          Esophageal Neoplasms Metabolism
          Cell Movement Physiology
          Esophageal Neoplasms
          Dose-Response Relationship, Drug
          Antineoplastic Agents Pharmacodynamics
          Cell Line, Tumor
          Cell Physiology Physiology
          Phosphorylation Physiology
          Human
      ab: Background: The MET receptor is a critical member of cancer-associated receptor tyrosine kinases and plays an important role in different biological activities, including differentiation, migration, and cell proliferation.Methods: In this study, novel MET inhibitors were introduced and applied on esophageal squamous carcinoma cell line KYSE-30, and the level of proliferation and migration, as well as the activated form of MET receptor protein were assessed in the examined cells. The human KYSE-30 cell line was cultured according to ATCC recommendations. The mRNA level of the MET gene was measured in the examined cell line using the quantitative RT-PCR assay. Cytotoxicity evaluation test was performed at different concentrations of heterocyclic anti-MET compounds (i.e. D1, D2, D5, D6, D7, and D8). Finally, the capability of these compounds in MET receptor inhibition was evaluated using the migration assay and Western blot. All experiments were performed in triplicate and repeated three times with similar results.Results: Cell growth and proliferation were significantly inhibited (p ≤ 0.05) by all the above-mentioned compounds. Moreover, the majority of compounds significantly prevented the cell migration (p ≤ 0.05) and inhibited MET autophosphorylation. Interestingly, the level of phosphorylated MET was significantly correlated with KYSE-30 cell migration.Conclusion: The obtained data introduced and confirmed the biological activities of the mentioned novel compounds in KYSE-30 cells and proposed that the therapeutic inhibition of MET with these compounds may be a powerful approach for inhibiting cancer cell migration and proliferation although some structural optimizations are needed to improve their inhibitory functions.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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