Orbitofrontal cortex thickness and substance use disorders in emerging adulthood: causal inferences from a co‐twin control/discordant twin study.

Background/aims: Research linking orbitofrontal cortex (OFC) structure and substance use disorders (SUDs) is largely correlational and often implies a causal effect of addiction/substance exposure on the brain, but familial risk factors (e.g. genetic liability) may confound these associations. We te...

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Publicado en:Addiction Vol. 116; no. 9; pp. 2548 - 2559
Autores principales: Harper, Jeremy, Wilson, Sylia, Malone, Stephen M., Hunt, Ruskin H., Thomas, Kathleen M., Iacono, William G.
Formato: research tables/charts Journal Article
Publicado: Wiley-Blackwell Sep2021
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Sep2021
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Orbitofrontal cortex thickness and substance use disorders in emerging adulthood: causal inferences from a co‐twin control/discordant twin study.
      aug:
        au:
          Harper, Jeremy
          Wilson, Sylia
          Malone, Stephen M.
          Hunt, Ruskin H.
          Thomas, Kathleen M.
          Iacono, William G.
        affil: Department of Psychiatry and Behavioral Sciences, University of Minnesota, Minneapolis MN,, USA
      sug:
        subj:
          Brain Anatomy and Histology
          Prefrontal Cortex Pathology
          Brain Cortical Thickness
          Substance Use Disorders In Adulthood
          Twins
          Human
          Causal Attribution
          Disease Susceptibility Familial and Genetic
          Adult
          Ethanol
          Cannabis
          Tobacco
          Clinical Assessment Tools
          Magnetic Resonance Imaging
          Comparative Studies
          Adult: 19-44 years
      ab: Background/aims: Research linking orbitofrontal cortex (OFC) structure and substance use disorders (SUDs) is largely correlational and often implies a causal effect of addiction/substance exposure on the brain, but familial risk factors (e.g. genetic liability) may confound these associations. We tested whether associations between alcohol, cannabis and tobacco use disorders and OFC thickness reflected the potential causal effects of familial risk or SUDs‐related consequences (e.g. substance exposure). Design A co‐twin control/discordant twin design separated familial risk confounding from SUD‐related consequences. Setting/participants: A population‐based sample of 436 24‐year‐old twins (62% monozygotic) from the Minnesota Twin Family Study, USA. Measurements Alcohol, cannabis and tobacco use disorders were assessed using the Composite International Diagnostic Interview—Substance Abuse Module. Cortical thickness of the medial and lateral OFC (mOFC and lOFC, respectively) was assessed using magnetic resonance imaging (MRI). Findings Lower mOFC (P‐values ≤ 0.006) but not lOFC (P‐values ≥ 0.190) thickness was observed in diagnosed individuals (n = 185) relative to non‐SUD controls (n = 251). Co‐twin control analyses offered evidence that mOFC associations were consistent with familial risk across SUDs (between‐pair effect: P‐values ≤ 0.047) and the independent consequences of having an alcohol or cannabis use disorder (within‐pair effect: P‐values ≤ 0.024). That is, within alcohol/cannabis discordant twin pairs, affected twins had significantly lower mOFC thickness compared with their unaffected co‐twins. Conclusions: A confounder‐adjusted analysis of the Minnesota Twin Family Study appeared to indicate that, beyond a substance use disorders general familial risk effect, the experience of an alcohol or cannabis use disorder in emerging adulthood reduces the thickness of the medial orbitofrontal cortex, a region associated with value‐guided decision‐making.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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