Influence of miRNA-30a-5p on Pulmonary Fibrosis in Mice with Streptococcus pneumoniae Infection through Regulation of Autophagy by Beclin-1.

The study is aimed at observing the influence of microribonucleic acid- (miRNA-) 30a-50p on the pulmonary fibrosis in mice with Streptococcus pneumoniae infection through the regulation of autophagy by Beclin-1. Specific pathogen-free mice were instilled with Streptococcus pneumoniae through the tra...

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Published in:BioMed Research International pp. 1 - 9
Main Authors: Liu, Hanyu, Li, Yabo, Zou, Yingdong, Zhang, Xingzong, Shi, Xiongfei, Yin, Zhiping, Lin, Yun
Format: pictorial research tables/charts Journal Article
Published: Wiley-Blackwell 9/24/2021
Online Access:View this record in EBSCOhost
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      dt: 9/24/2021
      pid: 480
      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2021/9963700
        152626625
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        atl: Influence of miRNA-30a-5p on Pulmonary Fibrosis in Mice with Streptococcus pneumoniae Infection through Regulation of Autophagy by Beclin-1.
      aug:
        au:
          Liu, Hanyu
          Li, Yabo
          Zou, Yingdong
          Zhang, Xingzong
          Shi, Xiongfei
          Yin, Zhiping
          Lin, Yun
        affil: Department of Laboratory, Yunnan Provincial Hospital of Traditional Chinese Medicine, Kunming, Yunnan 650021, China
      sug:
        subj:
          MicroRNA
          Pulmonary Fibrosis
          Streptococcus
          Beclin-1 Protein
          Animal Studies
          Mice
          Tumor Necrosis Factor Analysis
          Interleukins Analysis
          Oxidoreductases Analysis
          Enzyme-Linked Immunosorbent Assay
          Respiratory Function Tests
          Airway Pressure
          Gene Expression
          Polymerase Chain Reaction
          Blotting, Western
          Aminotransferases Blood
          Alanine Aminotransferase Blood
          Alkaline Phosphatase Blood
      ab: The study is aimed at observing the influence of microribonucleic acid- (miRNA-) 30a-50p on the pulmonary fibrosis in mice with Streptococcus pneumoniae infection through the regulation of autophagy by Beclin-1. Specific pathogen-free mice were instilled with Streptococcus pneumoniae through the trachea to establish the pulmonary fibrosis model. Then, they were divided into the miRNA-30a-50p mimics group (mimics group, n = 10) and miRNA-30a-5p inhibitors group (inhibitors group, n = 10), with the control group (n = 10) also set. Pulmonary tissue wet weight/dry weight (W/D) was detected. The content of tumor necrosis factor-α (TNF-α), interleukin- (IL-) 6, and myeloperoxidase (MPO) was determined using enzyme-linked immunosorbent assay (ELISA). Besides, the changes in the pulmonary function index dynamic lung compliance (Cdyn), plateau pressure (Pplat), and peak airway pressure (Ppeak) were monitored, and the gene and protein expression levels were measured via quantitative PCR (qPCR) and Western blotting. The expression level of miRNA-30a-5p was substantially raised in the mimics group (p < 0.05), but extremely low in the inhibitors group (p < 0.05). The mimics group had obviously raised levels of serum aminotransferase (AST), glutamic-pyruvic transaminase (GPT), alkaline phosphatase (ALP), and pulmonary tissue W/D (p < 0.05). Additionally, the expression levels of TNF-α, IL-6, and MPO were notably elevated in the mimics group, while their expression levels showed the opposite conditions in the inhibitors group (p < 0.05). According to the HE staining results, the inhibitors group had arranged orderly cells, while the mimics group exhibited lung injury, pulmonary edema, severe inflammatory response, and alveolar congestion. In the inhibitors group, Cdyn was remarkably elevated, but Pplat and Ppeak declined considerably (p < 0.05). Besides, the inhibitors group exhibited elevated messenger RNA (mRNA) levels of Beclin-1 and LC3, lowered mRNA levels of α-SMA and p62, a raised protein level of Beclin-1, and a markedly decreased protein level of p62 (p < 0.05). Silencing miRNA-30a-5p expression can promote the expression of Beclin-1 to accelerate the occurrence of autophagy, thereby treating pulmonary fibrosis in mice with Streptococcus pneumoniae infection.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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