Dark Sweet Cherry (Prunus avium) Phenolics Enriched in Anthocyanins Induced Apoptosis in MDA-MB-453 Breast Cancer Cells through MAPK-Dependent Signaling and Reduced Invasion via Akt and PLCγ-1 Downregulation.

Dark sweet cherries (DSCs) are rich source of phenolics known to exert anticancer and anti-invasive activities. This study elucidated the molecular mechanisms underlying the activity of DSC phenolics against MDA-MB-453 breast cancer cells In Vitro. Cells were treated with DSC phenolics in whole extr...

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Publicado en:Nutrition & Cancer Vol. 73; no. 10; pp. 1985 - 1998
Autores principales: Layosa, Marjorie Anne A., Lage, Nara N., Chew, Boon P., Atienza, Liezl, Mertens-Talcott, Susanne, Talcott, Stephen, Noratto, Giuliana D.
Formato: pictorial research tables/charts Journal Article
Publicado: Taylor & Francis Ltd 2021
Acceso en línea:Ver este registro en EBSCOhost
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        10.1080/01635581.2020.1817514
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        atl: Dark Sweet Cherry (Prunus avium) Phenolics Enriched in Anthocyanins Induced Apoptosis in MDA-MB-453 Breast Cancer Cells through MAPK-Dependent Signaling and Reduced Invasion via Akt and PLCγ-1 Downregulation.
      aug:
        au:
          Layosa, Marjorie Anne A.
          Lage, Nara N.
          Chew, Boon P.
          Atienza, Liezl
          Mertens-Talcott, Susanne
          Talcott, Stephen
          Noratto, Giuliana D.
        affil: Department of Nutrition and Food Science, Texas A&M University, College Station, TX, USA
      sug:
        subj:
          Breast Neoplasms Drug Therapy
          Wild Cherry Pharmacodynamics
          Phenols Pharmacodynamics
          Flavonoids Pharmacodynamics
          Apoptosis Drug Effects
          Cell Line, Tumor Drug Effects
          Transferases Drug Effects
          Signal Transduction Drug Effects
          Protein Kinases Drug Effects
          Peptides Drug Effects
          Human
          In Vitro Studies
          Growth Inhibitors
          Cell Movement Drug Effects
          Caspases Drug Effects
          Mitogen-Activated Protein Kinases Drug Effects
      ab: Dark sweet cherries (DSCs) are rich source of phenolics known to exert anticancer and anti-invasive activities. This study elucidated the molecular mechanisms underlying the activity of DSC phenolics against MDA-MB-453 breast cancer cells In Vitro. Cells were treated with DSC phenolics in whole extract (WE), and fractions enriched in anthocyanins (ACN) and proanthocyanidins (PCN) at concentrations that inhibited cell growth by 50%. Results showed that DSC phenolics suppressed Akt and PLCγ-1 activation, and inhibited cell motility and invasion, but only ACN reached significance. The extrinsic and intrinsic apoptotic pathways were also activated by DSC phenolics via caspase-8 cleavage and increased Bax/Bcl-2 ratio, with ACN exhibiting significant activation and stronger PARP-1 cleavage. Furthermore, sustained activation of mitogen-activated protein kinases (MAPKs) ERK1/2 and p38 was observed wherein ERK1/2 (U0126) and p38 (SB203580) inhibitors confirmed crosstalk ERK1/2-Akt and MAPK intrinsic mitochondrial pathways. In conclusion, DSC phenolics inhibited MDA-MB-453 breast cancer cells by targeting cell signaling pathways that induce apoptosis and suppress cell invasion, with ACN showing enhanced chemopreventive activities.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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