Impact of Fetal Growth Restriction on the Neonatal Microglial Proteome in the Rat.

Microglial activation is a key modulator of brain vulnerability in response to intra-uterine growth restriction (IUGR). However, the consequences of IUGR on microglial development and the microglial proteome are still unknown. We used a model of IUGR induced by a gestational low-protein diet (LPD) i...

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Publicado en:Nutrients Vol. 13; no. 11; pp. 3719 - 3720
Autores principales: Zinni, Manuela, Pansiot, Julien, Colella, Marina, Faivre, Valérie, Delahaye-Duriez, Andrée, Guillonneau, François, Bruce, Johanna, Salnot, Virginie, Mairesse, Jérôme, Knoop, Marit, Possovre, Marie-Laure, Vaiman, Daniel, Baud, Olivier
Formato: pictorial research tables/charts Journal Article
Publicado: MDPI Nov2021
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Nov2021
      vid: 13
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      pub: MDPI
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        atl: Impact of Fetal Growth Restriction on the Neonatal Microglial Proteome in the Rat.
      aug:
        au:
          Zinni, Manuela
          Pansiot, Julien
          Colella, Marina
          Faivre, Valérie
          Delahaye-Duriez, Andrée
          Guillonneau, François
          Bruce, Johanna
          Salnot, Virginie
          Mairesse, Jérôme
          Knoop, Marit
          Possovre, Marie-Laure
          Vaiman, Daniel
          Baud, Olivier
        affil: Faculté de Médecine, Inserm UMR 1141 NeuroDiderot, Université de Paris, F-75019 Paris, France
      sug:
        subj:
          Fetal Growth Retardation
          Glial Cells Metabolism
          Proteomics
          Dietary Fats Adverse Effects
          Animal Studies
          Rats
          Models, Biological
          Gene Expression
          Inflammation
          Immune System
          Oxidative Stress
          Reactive Oxygen Species
          Proteins Metabolism
          DNA Physiology
      ab: Microglial activation is a key modulator of brain vulnerability in response to intra-uterine growth restriction (IUGR). However, the consequences of IUGR on microglial development and the microglial proteome are still unknown. We used a model of IUGR induced by a gestational low-protein diet (LPD) in rats. Microglia, isolated from control and growth-restricted animals at P1 and P4, showed significant changes in the proteome between the two groups. The expression of protein sets associated with fetal growth, inflammation, and the immune response were significantly enriched in LPD microglia at P1 and P4. Interestingly, upregulation of protein sets associated with the oxidative stress response and reactive oxygen species production was observed at P4 but not P1. During development, inflammation-associated proteins were upregulated between P1 and P4 in both control and LPD microglia. By contrast, proteins associated with DNA repair and senescence pathways were upregulated in only LPD microglia. Similarly, protein sets involved in protein retrograde transport were significantly downregulated in only LPD microglia. Overall, these data demonstrate significant and multiple effects of LPD-induced IUGR on the developmental program of microglial cells, leading to an abnormal proteome within the first postnatal days.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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