hsa_circ_0023305 Enhances Laryngeal Squamous Cell Carcinoma Progression and Modulates TRPM7 via miR-218-5p Sponging.

Recently, circular RNAs have been shown to function as critical regulators of many human cancers. However, the circRNA mechanism in laryngeal squamous cell carcinoma (LSCC) remains elusive. Recent investigations using bioinformatics analysis revealed high expression of hsa_circ_0023305 in LSCC tissu...

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Publicado en:BioMed Research International pp. 1 - 12
Autores principales: Zhang, Yi, Tian, Kaisai, Zhou, Enhui, Xue, Xiaocheng, Yan, Shiling, Chen, Yixin, Qiao, Peipei, Yang, Liyun, Chen, Xiaoping
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 12/2/2021
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 12/2/2021
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2021/9968499
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        atl: hsa_circ_0023305 Enhances Laryngeal Squamous Cell Carcinoma Progression and Modulates TRPM7 via miR-218-5p Sponging.
      aug:
        au:
          Zhang, Yi
          Tian, Kaisai
          Zhou, Enhui
          Xue, Xiaocheng
          Yan, Shiling
          Chen, Yixin
          Qiao, Peipei
          Yang, Liyun
          Chen, Xiaoping
        affil: Department of Otorhinolaryngology Head and Neck Surgery, Shanghai Pudong New Area Gongli Hospital, Shanghai 200135, China
      sug:
        subj:
          MicroRNA Metabolism
          RNA, Circular Metabolism
          Laryngeal Neoplasms
          Neoplasms, Squamous Cell
          Disease Progression
          Bioinformatics
          Human
          Gene Expression
          Neoplasm Metastasis
          Lymph Nodes Pathology
          Neoplasm Invasiveness
          Cell Proliferation
          Cell Migration Inhibition
          Cell Line, Tumor
          In Vitro Studies
          In Vivo Studies
          China
      ab: Recently, circular RNAs have been shown to function as critical regulators of many human cancers. However, the circRNA mechanism in laryngeal squamous cell carcinoma (LSCC) remains elusive. Recent investigations using bioinformatics analysis revealed high expression of hsa_circ_0023305 in LSCC tissues compared to normal tissues. Furthermore, we discovered that hsa_circ_0023305 expression level was positively correlated to tumor/node/metastasis (TNM) stage as well as lymph node metastasis in LSCC. Moreover, higher hsa_circ_0023305 levels were correlated to poorer LSCC patient outcomes. Knockdown of hsa_circ_0023305 significantly inhibited LSCC cell proliferation, invasion, and migration abilities. Our team validated that hsa_circ_0023305 functioned as a miR-218-5p sponge from a mechanistic perspective, targeting the melastatin-related transient receptor potential 7 (TRPM7) in LSCC cells. TRPM7 regulates a nonselective cation channel and promotes cancer proliferation and metastasis. Our data demonstrated that miR-218-5p was downregulated in LSCC and that miR-218-5p upregulation repressed LSCC proliferation and invasion both in vivo and in vitro. Additionally, we found that hsa_circ_0023305-mediated upregulation of TRPM7 inhibited miR-218-5p and contributed to LSCC migration, proliferation, and invasion. In summary, these data propose a new mechanism by which the hsa_circ_0023305/miR-218-5p/TRPM7 network enhances LSCC progression.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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