Evaluation of renal fibrosis in various causes of glomerulonephritis by MR elastography: a clinicopathologic comparative analysis.

Background: Renal parenchymal fibrosis is the most important determinant of kidney disease progression and it is determined via biopsy. The aim of this study is to evaluate the renal stiffness noninvasively by magnetic resonance elastography (MRE) and to compare it with clinicopathologic parameters...

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Published in:Abdominal Radiology Vol. 47; no. 1; pp. 288 - 297
Main Authors: Güven, Alper Tuna, Idilman, Ilkay S., Cebrayilov, Cebrayil, Önal, Ceren, Kibar, Müge Üzerk, Sağlam, Arzu, Yıldırım, Tolga, Yılmaz, Rahmi, Altun, Bülent, Erdem, Yunus, Karçaaltıncaba, Muşturay, Arıcı, Mustafa
Format: diagnostic images pictorial research tables/charts Journal Article
Published: Springer Nature Jan2022
Online Access:View this record in EBSCOhost
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      dt: Jan2022
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00261-021-03296-1
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        atl: Evaluation of renal fibrosis in various causes of glomerulonephritis by MR elastography: a clinicopathologic comparative analysis.
      aug:
        au:
          Güven, Alper Tuna
          Idilman, Ilkay S.
          Cebrayilov, Cebrayil
          Önal, Ceren
          Kibar, Müge Üzerk
          Sağlam, Arzu
          Yıldırım, Tolga
          Yılmaz, Rahmi
          Altun, Bülent
          Erdem, Yunus
          Karçaaltıncaba, Muşturay
          Arıcı, Mustafa
        affil: Department of Internal Medicine, Faculty of Medicine, Hacettepe University, Ankara, Turkey
      sug:
        subj:
          Glomerulonephritis
          Kidney Diseases
          Fibrosis
          Ultrasonography Methods
          Magnetic Resonance Imaging Methods
          Human
          Comparative Studies
          Descriptive Statistics
          Data Analysis Software
          Male
          Female
          Amyloidosis
          Mann-Whitney U Test
          Spearman's Rank Correlation Coefficient
          Male
          Female
      ab: Background: Renal parenchymal fibrosis is the most important determinant of kidney disease progression and it is determined via biopsy. The aim of this study is to evaluate the renal stiffness noninvasively by magnetic resonance elastography (MRE) and to compare it with clinicopathologic parameters in glomerulonephritis and AA amyloidosis patients. Methods: Thirty-four patients with glomerular filtration rate (GFR) over 20 ml/min/1.73m2 had non-contrast MRE prospectively. Kidney stiffness values were obtained from whole kidney, cortex, and medulla. Values were correlated with GFR, albuminuria, proteinuria, and degree of fibrosis that are assessed via renal biopsy. Patients were grouped clinicopathologically to assess the relation between stiffness and chronicity. Results: Mean whole kidney, cortex, and medulla stiffnesses were 3.78 (± 1.26), 3.63 (± 1.25), and 4.77 (± 2.03) kPa, respectively. Mean global glomerulosclerosis was 22% (± 18%) and median segmental glomerulosclerosis was 4% (min–max: 0%–100%). Extent of tubulointerstitial fibrosis was less than 25% in 26 of the patients (76.5%), 25%–50% in 6 of the patients (17.6%), and higher than 50% in 2 of the patients (5.9%). Fourteen patients were defined to have chronic renal parenchymal injury. MRE-derived stiffness values correlated negatively with parameters of fibrosis. Lower stiffness values were observed in patients with chronic renal injury compared to those without (P < 0.05 for whole kidney and medulla MRE-derived stiffness). Conclusion: MRE-derived stiffness values were lower in patients with chronic injury. Stiffness decreases as glomerulosclerosis and tubulointerstitial fibrosis progresses in patients with primary glomerulonephritis and AA amyloidosis. With future studies, there may be a role for MRE to assess renal function in concert with conventional markers.
      pubtype: Academic Journal
      doctype:
        diagnostic images
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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