Nicotinic Acid Riboside Regulates Nrf-2/P62-Related Oxidative Stress and Autophagy to Attenuate Doxorubicin-Induced Cardiomyocyte Injury.

Doxorubicin (Dox) is an effective chemotherapeutic drug for the treatment of various cancers. Due to its potential fatal cardiotoxic side effects, the clinical application is often limited. Dexrazoxane (Dex) is the only drug approved by the Food and Drug Administration (FDA) for the prevention of Do...

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Publicado en:BioMed Research International pp. 1 - 15
Autores principales: Zou, Linfeng, Liang, Bing, Gao, YuanZhen, Ye, Ting, Li, MengJiao, Zhang, Yukun, Lu, Qi, Hu, Xiaokun, Li, Huanting, Yuan, Yang, Xing, Dongming
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 2/22/2022
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2/22/2022
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      pub: Wiley-Blackwell
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        10.1155/2022/6293329
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        atl: Nicotinic Acid Riboside Regulates Nrf-2/P62-Related Oxidative Stress and Autophagy to Attenuate Doxorubicin-Induced Cardiomyocyte Injury.
      aug:
        au:
          Zou, Linfeng
          Liang, Bing
          Gao, YuanZhen
          Ye, Ting
          Li, MengJiao
          Zhang, Yukun
          Lu, Qi
          Hu, Xiaokun
          Li, Huanting
          Yuan, Yang
          Xing, Dongming
        affil: School of Basic Medicine and the Affiliated Hospital of Qingdao University, Qingdao University, Qingdao 266071, China
      sug:
        subj:
          Doxorubicin Adverse Effects
          Cardiotoxicity Prevention and Control
          Niacin Pharmacodynamics
          Oxidative Stress Drug Effects
          Autophagy Drug Effects
          Myocytes, Cardiac Drug Effects
          NF-E2-Related Factor 2 Drug Effects
          Animal Studies
          Mice
          In Vivo Studies
          In Vitro Studies
          Troponin Drug Effects
          Heart Drug Effects
          Ventricular Ejection Fraction Drug Effects
          Malondialdehyde Drug Effects
          Lactate Dehydrogenase Drug Effects
          Reactive Oxygen Species Drug Effects
          Superoxide Dismutase Drug Effects
          Glutathione Drug Effects
          Cell Viability Drug Effects
          Apoptosis Drug Effects
          Treatment Outcomes
      ab: Doxorubicin (Dox) is an effective chemotherapeutic drug for the treatment of various cancers. Due to its potential fatal cardiotoxic side effects, the clinical application is often limited. Dexrazoxane (Dex) is the only drug approved by the Food and Drug Administration (FDA) for the prevention of Dox-induced cardiotoxicity but has side effects. Thus, more protective strategies should be explored. If NAD+ plays a role in maintaining heart function, its precursor prospectively alleviates Dox-induced cellular injury. Here, we studied the protective effects of nicotinic acid riboside (NAR) on Dox-induced cardiotoxicity in vivo and in vitro. We found that NAR significantly improved the cardiac function of Dox-treated mice by restoring ejection fraction (EF), fractional shortening (FS), and serum level of cardiac troponin (cTnI). NAR not only reduced malondialdehyde (MDA), lactate dehydrogenase (LDH), and reactive oxygen species (ROS) levels in Dox-treated cardiomyocytes but also further promoted the activities of cardiac superoxide dismutase (SOD) and glutathione (GSH). Following exposure to 5 μM Dox, cotreatment with NAR exhibited increased cell viability with a decrease in the apoptosis cell population. Moreover, the levels of apoptosis-related proteins, as well as proteins involved in oxidative stress and autophagy, were altered after NAR treatment. Collectively, these findings underline the protective potential of NAR against Dox-induced cardiomyocyte injury by regulating Nrf-2/P62-related oxidative stress and autophagy, which could potentially promote survival.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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