Investigation of the relationship between community-acquired respiratory distress syndrome toxin and the high-mobility group box protein 1-toll-like receptors-myeloid differentiation factor 88 signaling pathway in Mycoplasma pneumoniae pneumonia.

Background: In recent years, reports of refractory Mycoplasma pneumoniae pneumonia (RMPP) have gradually increased, including reports on how these conditions threaten the lives of children. However, the specific mechanism of Mycoplasma pneumoniae pneumonia (MPP) remains unclear. This study aimed to...

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Publicado en:Italian Journal of Pediatrics Vol. 48; no. 1; pp. 1 - 7
Autores principales: Fan, Yujie, Ding, Ying, Li, Yuqin, Zhang, Dandan, Yu, Min, Zhou, Wei-fang, Kong, Xiaoxing
Formato: research tables/charts Journal Article
Publicado: BioMed Central 5/3/2022
Acceso en línea:Ver este registro en EBSCOhost
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        17208424
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      jtl: Italian Journal of Pediatrics
      issn: 17208424
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      dt: 5/3/2022
      vid: 48
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      pub: BioMed Central
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        156643073
        156643073
        156643073
        10.1186/s13052-022-01254-1
        156643073
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        atl: Investigation of the relationship between community-acquired respiratory distress syndrome toxin and the high-mobility group box protein 1-toll-like receptors-myeloid differentiation factor 88 signaling pathway in Mycoplasma pneumoniae pneumonia.
      aug:
        au:
          Fan, Yujie
          Ding, Ying
          Li, Yuqin
          Zhang, Dandan
          Yu, Min
          Zhou, Wei-fang
          Kong, Xiaoxing
        affil: Department of Infectious Disease, Children's Hospital of Soochow University, Soochow University, 215003, Suzhou, China
      sug:
        subj:
          Gene Expression
          Respiratory Distress Syndrome, Acute
          Bacterial Toxins
          Bacterial Proteins
          Pneumonia, Mycoplasma
          Toll-Like Receptors
          Receptors, Cell Surface
          Signal Transduction
          Human
          Bronchoscopy
          Descriptive Statistics
          Male
          Female
          Child
          Child, Preschool
          Pneumonia
          Signs and Symptoms
          Polymerase Chain Reaction
          Quantitative Studies
          Comparative Studies
          Tumor Necrosis Factor
          Enzyme-Linked Immunosorbent Assay
          Data Analysis Software
          T-Tests
          Pearson's Correlation Coefficient
          Correlation Coefficient
          Chi Square Test
          Child: 6-12 years
          Child, Preschool: 2-5 years
          Male
          Female
      ab: Background: In recent years, reports of refractory Mycoplasma pneumoniae pneumonia (RMPP) have gradually increased, including reports on how these conditions threaten the lives of children. However, the specific mechanism of Mycoplasma pneumoniae pneumonia (MPP) remains unclear. This study aimed to investigate the relationship between community-acquired respiratory distress syndrome toxin (CARDS TX) and High-mobility group box protein 1-Toll-like receptors-Myeloid differentiation factor 88 (HMGB1-TLRs-MyD88) in MPP and to examine the immune pathogenesis of Mycoplasma pneumoniae infection. Methods: Children who were diagnosed with MPP and examined by bronchoscopy were included in the MPP group. Additionally, children who underwent bronchoscopy because of bronchial foreign bodies in the same period were included in the control group. Gene expression of CARDS TX, HMGB1, Toll-like receptor 2 (TLR2), Toll-like receptor 4 (TLR4), MyD88, and cluster of differentiation 14 (CD14) in bronchoalveolar lavage fluid (BALF) were detected using real-time reverse transcription-polymerase chain reaction. Correlations between CARDS TX and HMGB1-TLRs-MyD88 were analyzed. Results: CARDS TX, HMGB1, TLR2, MyD88, and CD14 mRNA expression in BALF in the MPP group was significantly higher than that in the control group (all P < 0.05). CARDS TX mRNA expression was positively correlated with HMGB1, TLR2, MyD88, and CD14 mRNA expression (all P < 0.05). Furthermore, HMGB1 mRNA expression was positively correlated with TLR2, MyD88, and CD14 mRNA expression (all P < 0.05). Conclusions: CARDS TX may participate in the immune pathogenesis of MPP through the HMGB1-TLRs/CD14-MyD88 pathway.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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