Enhanced Association of Tau Pathology and Cognitive Impairment in Mild Cognitive Impairment Subjects with Behavior Symptoms.

Background: Mild cognitive impairment (MCI) individuals with neuropsychiatric symptoms (NPS) are more likely to develop dementia.Objective: We sought to understand the relationship between neuroimaging markers such as tau pathology and cognitive symptoms both with and without the presence of NPS dur...

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Publicado en:Journal of Alzheimer's Disease Vol. 87; no. 2; pp. 557 - 569
Autores principales: Ge, Xinting, Qiao, Yuchuan, Choi, Jiyoon, Raman, Rema, Ringman, John M., Shi, Yonggang
Formato: research Journal Article
Publicado: Sage Publications Inc. 2022
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2022
      vid: 87
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      pub: Sage Publications Inc.
      place: Thousand Oaks, California
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        157146106
        157146106
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        157146106
        10.3233/JAD-215555
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        157146106
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      tig:
        atl: Enhanced Association of Tau Pathology and Cognitive Impairment in Mild Cognitive Impairment Subjects with Behavior Symptoms.
      aug:
        au:
          Ge, Xinting
          Qiao, Yuchuan
          Choi, Jiyoon
          Raman, Rema
          Ringman, John M.
          Shi, Yonggang
        affil: Laboratory of Neuro Imaging (LONI), Stevens Neuroimaging and Informatics Institute, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA
      sug:
        subj:
          Alzheimer's Disease Pathology
          Human
          Nerve Tissue Proteins
          Peptides
          Tomography, Emission-Computed Methods
          Comparative Studies
          Multicenter Studies
          Evaluation Research
          Validation Studies
      ab: Background: Mild cognitive impairment (MCI) individuals with neuropsychiatric symptoms (NPS) are more likely to develop dementia.Objective: We sought to understand the relationship between neuroimaging markers such as tau pathology and cognitive symptoms both with and without the presence of NPS during the prodromal period of Alzheimer's disease.Methods: A total of 151 MCI subjects with tau positron emission tomographic (PET) scanning with 18F AV-1451, amyloid-β (Aβ) PET scanning with florbetapir or florbetaben, magnetic resonance imaging, and cognitive and behavioral evaluations were selected from the Alzheimer's Disease Neuroimaging Initiative. A 4-group division approach was proposed using amyloid (A-/A+) and behavior (B-/B+) status: A-B-, A-B+, A+B-, and A+B+. Pearson's correlation test was conducted for each group to examine the association between tau deposition and cognitive performance.Results: No statistically significant association between tau deposition and cognitive impairment was found for subjects without behavior symptoms in either the A-B-or A+B-groups after correction for false discovery rate. In contrast, tau deposition was found to be significantly associated with cognitive impairment in entorhinal cortex and temporal pole for the A-B+ group and nearly the whole cerebrum for the A+B+ group.Conclusion: Enhanced associations between tauopathy and cognitive impairment are present in MCI subjects with behavior symptoms, which is more prominent in the presence of elevated amyloid pathology. MCI individuals with NPS may thus be at greater risk for further cognitive decline with the increase of tau deposition in comparison to those without NPS.
      pubtype: Academic Journal
      doctype:
        research
        Journal Article
      ougenre: Article
    language: English
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