Influence of a proinflammatory state on postprandial outcomes in elderly subjects with a risk phenotype for cardiometabolic diseases.

Purpose: Low-grade inflammation in obesity is associated with insulin resistance and other metabolic disturbances. In response to high-energy meal intake, blood concentrations of inflammatory markers, glucose and insulin rise. The aim of this study was to examine whether a basal inflammatory state i...

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Publicado en:European Journal of Nutrition Vol. 61; no. 6; pp. 3077 - 3084
Autores principales: Schönknecht, Yannik Bernd, Crommen, Silke, Stoffel-Wagner, Birgit, Coenen, Martin, Fimmers, Rolf, Stehle, Peter, Egert, Sarah
Formato: research tables/charts Journal Article
Publicado: Springer Nature Sep2022
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Sep2022
      vid: 61
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00394-022-02870-7
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        atl: Influence of a proinflammatory state on postprandial outcomes in elderly subjects with a risk phenotype for cardiometabolic diseases.
      aug:
        au:
          Schönknecht, Yannik Bernd
          Crommen, Silke
          Stoffel-Wagner, Birgit
          Coenen, Martin
          Fimmers, Rolf
          Stehle, Peter
          Egert, Sarah
        affil: Department of Nutrition and Food Science, Nutritional Physiology, University of Bonn, Nussallee 9, 53115, Bonn, Germany
      sug:
        subj:
          Cardiometabolic Risk Factors
          Phenotype
          Inflammation
          Postprandial Period
          Metabolomics
          Human
          Crossover Design
          Diet
          Fasting
          Blood Glucose Analysis
          Insulin Analysis
          Interleukins Analysis
          Receptors, Cell Surface
          C-Reactive Protein
          Insulin Resistance
      ab: Purpose: Low-grade inflammation in obesity is associated with insulin resistance and other metabolic disturbances. In response to high-energy meal intake, blood concentrations of inflammatory markers, glucose and insulin rise. The aim of this study was to examine whether a basal inflammatory state influences postprandial responses. Methods: A randomized crossover trial was performed in 60 participants with a cardiometabolic risk phenotype (age 70 ± 5 years; BMI 30.9 ± 3.1 kg/m2). Each participant consumed three different iso-energetic meals (4300 kJ): a Western diet-like high-fat meal (WDHF), a Western diet-like high-carbohydrate meal (WDHC) and a Mediterranean diet-like meal (MED). Blood samples were collected when fasted and hourly for 5 h postprandially and analyzed for glucose, insulin, interleukin-1β (IL-1β), interleukin-6 (IL-6) and endothelial adhesion molecules. Based on fasting serum C-reactive protein (CRP) concentrations, participants were assigned to a high inflammation (CRP ≥ 2.0 mg/L; n = 30) or low inflammation (CRP < 2.0 mg/L; n = 30) group, and postprandial outcomes were compared. Results: Plasma IL-6, glucose and serum insulin increased after all meals, while IL-1β and endothelial adhesion molecules were unchanged. The high inflammation group had higher fasting and postprandial IL-6 concentrations than the low inflammation group, although the IL-6 response slope was similar between groups. In response to the WDHC meal, participants in the high inflammation group experienced a higher glycaemic response than those in the low inflammation group. Conclusion: A basal proinflammatory state results in higher absolute fasting and postprandial IL-6 concentrations, but the increase in IL-6 relative to basal levels is not different between high and low inflammation groups. Elevated glycaemic response in the high inflammation group may be due to inflammation-induced short-term insulin resistance. The trial was registered at http://www.germanctr.de and http://www.drks.de under identifier DRKS00009861 (registration date, January 22, 2016).
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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