The Effect of Osteoprotectin (OPG)/Receptor Activator of Nuclear Factor-κB Ligand (RANKL)/Receptor Activator of Nuclear Factor-κB (RANK) Gene Methylation on Aortic Valve Calcified.

To evaluate the effect of the methylation of osteoprotectin (OPG)/receptor activator of nuclear factor-κB ligand (RANKL)/receptor activator of nuclear factor-κB (RANK) pathway on aortic valve calcification, the aortic valve tissue was collected from 38 aortic stenosis (AS) patients who underwent val...

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Publicado en:BioMed Research International pp. 1 - 11
Autores principales: Luo, Wei, Song, Yanqiu, Wang, Jing, Yang, Xia, Li, Zuocheng, Cong, Hongliang
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 8/12/2022
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 8/12/2022
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        10.1155/2022/1592576
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        atl: The Effect of Osteoprotectin (OPG)/Receptor Activator of Nuclear Factor-κB Ligand (RANKL)/Receptor Activator of Nuclear Factor-κB (RANK) Gene Methylation on Aortic Valve Calcified.
      aug:
        au:
          Luo, Wei
          Song, Yanqiu
          Wang, Jing
          Yang, Xia
          Li, Zuocheng
          Cong, Hongliang
        affil: Department of Cardiovascular Disease, Tianjin Chest Hospital, Tianjin 300222, China
      sug:
        subj:
          Methylation
          Calcinosis
          Aortic Valve Pathology
          Tumor Necrosis Factor Pharmacodynamics
          Membrane Proteins Pharmacodynamics
          Aortic Valve Stenosis Surgery
          Treatment Outcomes
          Human
          Reverse Transcriptase Polymerase Chain Reaction
          Enzyme-Linked Immunosorbent Assay
          Staining and Labeling
          Immunohistochemistry
          RNA, Messenger
          Gene Expression
          Descriptive Statistics
      ab: To evaluate the effect of the methylation of osteoprotectin (OPG)/receptor activator of nuclear factor-κB ligand (RANKL)/receptor activator of nuclear factor-κB (RANK) pathway on aortic valve calcification, the aortic valve tissue was collected from 38 aortic stenosis (AS) patients who underwent valve replacement. OPG and RANKL gene methylation, RT-PCR, and ELISA were performed. Hematoxylin-eosin staining (HE), alizarin red-S staining, and immunohistochemically staining of OPG, RANKL, and CD68 were simultaneously performed. The patients were divided into noncalcified group (n = 21) and calcified group (n = 17). The methylation rate of OPG gene in noncalcified group was higher than that in calcified group (P = 0.027). The methylation degree of RANKL gene was generally lower, but the noncalcified group was still higher than that in the calcified group (P = 0.025). RT-PCR analysis showed that the mRNA expression of OPG and RANKL was higher in calcified group than in noncalcified group (P = 0.007 and P = 0.036 , respectively), and the mRNA expression was negatively correlated with the gene methylation rate. The protein expression of OPG and RANKL was detected by immunohistochemistry and ELISA, showing significantly increased in calcified group (P = 0.004 and P = 0.042 , respectively). Soluble RANKL (sRANKL) in CD68-positive group was significantly different from that in negative group (0.1243 ± 0.0321 vs 0.0984 ± 0.0218 pg/mL, P = 0.007). There was no significant difference in OPG value between positive group (1.9411 ± 0.4554 ng/mL) and negative group (1.8422 ± 0.5218 ng/mL, P = 0.587). In conclusion, the degree of methylation of OPG and RANKL genes may play an important role in regulating valve calcification in AS patients.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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