Dietary Xenobiotics Derived from Food Processing: Association with Fecal Mutagenicity and Gut Mucosal Damage.

Whereas the mechanisms underlying the association of toxic dietary xenobiotics and cancer risk are not well established, it is plausible that dietary pattern may affect the colon environment by enhancing or reducing exposure to mutagens. This work aimed to investigate the association between xenobio...

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Publicado en:Nutrients Vol. 14; no. 17; pp. 3482 - 3500
Autores principales: Ruiz-Saavedra, Sergio, Zapico, Aida, del Rey, Carmen González, Gonzalez, Celestino, Suárez, Adolfo, Díaz, Ylenia, de los Reyes-Gavilán, Clara G., González, Sonia
Formato: pictorial research tables/charts Journal Article
Publicado: MDPI Sep2022
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Sep2022
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        atl: Dietary Xenobiotics Derived from Food Processing: Association with Fecal Mutagenicity and Gut Mucosal Damage.
      aug:
        au:
          Ruiz-Saavedra, Sergio
          Zapico, Aida
          del Rey, Carmen González
          Gonzalez, Celestino
          Suárez, Adolfo
          Díaz, Ylenia
          de los Reyes-Gavilán, Clara G.
          González, Sonia
        affil: Department of Microbiology and Biochemistry of Dairy Products, Instituto de Productos Lácteos de Asturias (IPLA-CSIC), 33300 Villaviciosa, Spain
      sug:
        subj:
          Diet
          Xenobiotics
          Food Poisoning
          Feces Analysis
          Mutagens
          Intestinal Mucosa Physiopathology
          Colorectal Neoplasms Prevention and Control
          Human
          Male
          Middle Age
          Aged
          Prospective Studies
          Case Control Studies
          Intestinal Polyps
          Microscopy Utilization
          Carcinogens
          Ethanol
          Hydrocarbons
          Polycyclic Hydrocarbons, Aromatic
          Plants, Edible
          Heterocyclic Compounds
          Amines
          Colonoscopy
          Food Handling
          Pyridines
          Middle Aged: 45-64 years
          Aged: 65+ years
          Male
      ab: Whereas the mechanisms underlying the association of toxic dietary xenobiotics and cancer risk are not well established, it is plausible that dietary pattern may affect the colon environment by enhancing or reducing exposure to mutagens. This work aimed to investigate the association between xenobiotics intake and different stages of intestinal mucosal damage and colorectal cancer (CRC) screening and examine whether these associations may be mediated by altered intestinal mutagenicity. This was a case control study with 37 control subjects, 49 patients diagnosed with intestinal polyps, and 7 diagnosed with CRC. Lifestyle, dietary, and clinical information was registered after colonoscopy. For xenobiotics intake estimation the European Prospective Investigation into Cancer (EPIC) and the Computerized Heterocyclic Amines Resource for Research in Epidemiology of Disease (CHARRED) databases were used. The mutagenicity of fecal supernatants was assayed by the Ames test and light microscopy was used for the presence of aberrant crypt formation. Among all the potential carcinogens studied, the polyp group showed higher intakes of ethanol and dibenzo (a) anthracene (DiB(a)A). Besides, intakes between 0.75 and 1.29 µg/d of total polycyclic aromatic hydrocarbons (PAHs) were related with a higher risk of belonging to the polyp group. On the contrary, an intake of wholegrain cereals greater than 50 g/d was associated with a reduction in the relative risk of belonging to the polyp group. Heterocyclic amines (HAs) such as 2-amino-1-methyl-6-phenylimidazo (4,5,b) pyridine (PhIP) were associated with an increased level of mutagenicity in polyps. This study is of great interest for the identification of possible therapeutic targets for the early prevention of colon cancer through diet.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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