Anti-Inflammatory Effect of Korean Propolis on Helicobacter pylori -Infected Gastric Mucosal Injury Mice Model.

Propolis, a natural resinous substance obtained from a variety of buds and plants, has been reported to possess various biological functions. Several recent studies have demonstrated the inhibitory effects of propolis on the growth of Helicobacter pylori (H. pylori) in vitro; however, current resear...

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Publicado en:Nutrients Vol. 14; no. 21; pp. 4644 - 4659
Autores principales: Song, Moon-Young, Lee, Da-Young, Han, Young-Min, Kim, Eun-Hee
Formato: pictorial research tables/charts Journal Article
Publicado: MDPI Nov2022
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Nov2022
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      pub: MDPI
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        atl: Anti-Inflammatory Effect of Korean Propolis on Helicobacter pylori -Infected Gastric Mucosal Injury Mice Model.
      aug:
        au:
          Song, Moon-Young
          Lee, Da-Young
          Han, Young-Min
          Kim, Eun-Hee
        affil: College of Pharmacy and Institute of Pharmaceutical Sciences, CHA University, Seongnam 13488, Korea
      sug:
        subj:
          Antiinflammatory Agents Pharmacodynamics
          Propolis Pharmacodynamics
          Helicobacter Infections Drug Therapy
          Gastric Mucosa Injuries
          Gastric Mucosa Drug Effects
          Wounds and Injuries Drug Therapy
          Animal Studies
          Mice
          In Vitro Studies
          In Vivo Studies
          Cytotoxins
          Comparative Studies
          Gene Expression
          NF-kappa B
          South Korea
          Geographic Factors
          Treatment Outcomes
      ab: Propolis, a natural resinous substance obtained from a variety of buds and plants, has been reported to possess various biological functions. Several recent studies have demonstrated the inhibitory effects of propolis on the growth of Helicobacter pylori (H. pylori) in vitro; however, current research efforts on Korean propolis (KP) remain insufficient especially in vivo. Our study aims to investigate the anti-inflammatory effect and molecular mechanism of KP on mouse gastric mucosa during H. pylori infection. We examined an in vivo H. pylori-induced gastric mucosal injury mice model. We found that KP inhibited the growth of H. pylori and attenuated the expression of H. pylori virulence factors such as cytotoxin-associated gene A, encoding urease A subunit, surface antigen gene and neutrophil-activating protein A. Moreover, KP reduced both gross lesions and pathological scores in H. pylori-challenged mice. In addition, KP markedly restrained the production of pro-inflammatory cytokines and nitric oxide levels compared with an untreated H. pylori-infected group. In particular, we found that KP repressed the phosphorylation of IκBα and NF-κB p65 subunit, and subsequently suppressed their downstream target genes. Taken together, these findings demonstrate the beneficial effects of KP on inflammation through the inhibition of NF-κB signaling as well as inhibition of H. pylori growth in a mouse model infected with H. pylori. This suggests the potential application of KP as a natural supplement for patient's suffering from gastric mucosal injury caused by H. pylori infection.
      pubtype: Academic Journal
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        pictorial
        research
        tables/charts
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      ougenre: Article
    language: English
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