Resveratrol Suppresses Bupivacaine-Induced Spinal Neurotoxicity in Rats by Inhibiting Endoplasmic Reticulum Stress via SIRT1 Modulation.

Bupivacaine (BUP) may cause neurotoxic effects after spinal anesthesia. Resveratrol (RSV), a natural agonist of Silent information regulator 1 (SIRT1), protects various tissues and organs from damage by regulating endoplasmic reticulum (ER) stress. The aim of this study is to explore whether RSV cou...

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Publicado en:BioMed Research International pp. 1 - 14
Autores principales: Luo, Yunpeng, Zhao, Yang, Lai, Jian, Wei, Liling, Zhou, Gang, Yu, Yue, Liu, Jingchen
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell 2/21/2023
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 2/21/2023
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      pub: Wiley-Blackwell
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        10.1155/2023/1176232
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        atl: Resveratrol Suppresses Bupivacaine-Induced Spinal Neurotoxicity in Rats by Inhibiting Endoplasmic Reticulum Stress via SIRT1 Modulation.
      aug:
        au:
          Luo, Yunpeng
          Zhao, Yang
          Lai, Jian
          Wei, Liling
          Zhou, Gang
          Yu, Yue
          Liu, Jingchen
        affil: Department of Anesthesiology, The First Affiliated Hospital of Guangxi Medical University, 22 Shuangyong Road, Nanning, 530021 Guangxi, China
      sug:
        subj:
          Resveratrol Pharmacodynamics
          Bupivacaine Adverse Effects
          Neurotoxicity Syndromes Risk Factors
          Spinal Diseases Risk Factors
          Endoplasmic Reticulum Stress Drug Effects
          Oxidative Stress Drug Effects
          Transferases Metabolism
          Risk Assessment
          Anesthesia, Spinal Adverse Effects
          Animal Studies
          Rats
          Injections, Intraspinal
          Bupivacaine Administration and Dosage
          Behavior, Animal
          Staining and Labeling
          Histocytochemistry
          Apoptosis
          Neurons Pathology
          Gene Expression
          Fluorescent Antibody Technique
          Blotting, Western
          RNA, Messenger Metabolism
          Funding Source
      ab: Bupivacaine (BUP) may cause neurotoxic effects after spinal anesthesia. Resveratrol (RSV), a natural agonist of Silent information regulator 1 (SIRT1), protects various tissues and organs from damage by regulating endoplasmic reticulum (ER) stress. The aim of this study is to explore whether RSV could alleviate the neurotoxicity induced by bupivacaine via regulating ER stress. We established a model of bupivacaine-induced spinal neurotoxicity in rats using intrathecal injection of 5% bupivacaine. The protective effect of RSV was evaluated by injecting intrathecally with 30 μg/μL RSV in total of 10 μL per day for 4 consecutive days. On day 3 after bupivacaine administration, tail-flick latency (TFL) tests and the Basso, Beattie, and Bresnahan (BBB) locomotor scores were assessed to neurological function, and the lumbar enlargement of the spinal cord was obtained. H&E and Nissl staining were used to evaluate the histomorphological changes and the number of survival neurons. TUNEL staining was conducted to determine apoptotic cells. The expression of proteins was detected by IHC, immunofluorescence, and western blot. The mRNA level of SIRT1 was determined by RT-PCR. Bupivacaine caused spinal cord neurotoxicity by inducing cell apoptosis and triggering ER stress. RSV treatment promoted the recovery of neurological dysfunction after bupivacaine administration by suppressing neuronal apoptosis and ER stress. Furthermore, RSV upregulated SIRT1 expression and inhibited PERK signaling pathway activation. In summary, resveratrol suppresses bupivacaine-induced spinal neurotoxicity in rats by inhibiting endoplasmic reticulum stress via SIRT1 modulation.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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