Optimisation Models for Pathway Activity Inference in Cancer.

Simple Summary: Subtype classification and prognostic prediction are key research targets in complex diseases such as cancer. In this work, an optimisation model was designed to infer the activity of biological pathways from gene expression values. The optimisation model enables the pathway activity...

Full description

Bibliographic Details
Published in:Cancers Vol. 15; no. 6; pp. 1787 - 1806
Main Authors: Chen, Yongnan, Liu, Songsong, Papageorgiou, Lazaros G., Theofilatos, Konstantinos, Tsoka, Sophia
Format: equations & formulas research tables/charts Journal Article
Published: MDPI Mar2023
Online Access:View this record in EBSCOhost
fields @attributes:
  recordID: 1
pdfLink:
plink: https://search.ebscohost.com/login.aspx?direct=true&db=ccm&AN=162751186&site=ehost-live
header:
  @attributes:
    shortDbName: ccm
    uiTerm: 162751186
    longDbName: CINAHL Complete
    uiTag: AN
  controlInfo:
    bkinfo:
    dissinfo:
    jinfo:
      jid:
        20726694
        B74B
      jtl: Cancers
      issn: 20726694
      maglogo: N
    pubinfo:
      dt: Mar2023
      vid: 15
      iid: 6
      pid: 97109
      pub: MDPI
    artinfo:
      ui:
        162751186
        162751186
        162751186
        10.3390/cancers15061787
        162751186
      ppf: 1787
      ppct: 19
      formats:
      tig:
        atl: Optimisation Models for Pathway Activity Inference in Cancer.
      aug:
        au:
          Chen, Yongnan
          Liu, Songsong
          Papageorgiou, Lazaros G.
          Theofilatos, Konstantinos
          Tsoka, Sophia
        affil: Department of Informatics, Faculty of Natural, Mathematical and Engineering Sciences, King's College London, Bush House, London WC2B 4BG, UK
      sug:
        subj:
          Neoplasms Classification
          Models, Biological
          Gene Expression Profiling
          Signal Transduction
          Human
          Models, Theoretical
          Phenotype
          Sequence Analysis
          RNA
          Genes
          Breast Neoplasms
          Colorectal Neoplasms
          Survival Analysis
          Funding Source
      ab: Simple Summary: Subtype classification and prognostic prediction are key research targets in complex diseases such as cancer. In this work, an optimisation model was designed to infer the activity of biological pathways from gene expression values. The optimisation model enables the pathway activity values to separate the sample subtypes to the greatest extent, thereby improving sample classification accuracy. The proposed model was evaluated on cancer molecular subtype classification, robustness to noisy data and survival prediction, and allowed the identification of disease-important genes and pathways. Background: With advances in high-throughput technologies, there has been an enormous increase in data related to profiling the activity of molecules in disease. While such data provide more comprehensive information on cellular actions, their large volume and complexity pose difficulty in accurate classification of disease phenotypes. Therefore, novel modelling methods that can improve accuracy while offering interpretable means of analysis are required. Biological pathways can be used to incorporate a priori knowledge of biological interactions to decrease data dimensionality and increase the biological interpretability of machine learning models. Methodology: A mathematical optimisation model is proposed for pathway activity inference towards precise disease phenotype prediction and is applied to RNA-Seq datasets. The model is based on mixed-integer linear programming (MILP) mathematical optimisation principles and infers pathway activity as the linear combination of pathway member gene expression, multiplying expression values with model-determined gene weights that are optimised to maximise discrimination of phenotype classes and minimise incorrect sample allocation. Results: The model is evaluated on the transcriptome of breast and colorectal cancer, and exhibits solution results of good optimality as well as good prediction performance on related cancer subtypes. Two baseline pathway activity inference methods and three advanced methods are used for comparison. Sample prediction accuracy, robustness against noise expression data, and survival analysis suggest competitive prediction performance of our model while providing interpretability and insight on key pathways and genes. Overall, our work demonstrates that the flexible nature of mathematical programming lends itself well to developing efficient computational strategies for pathway activity inference and disease subtype prediction.
      pubtype: Academic Journal
      doctype:
        equations & formulas
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
    refInfo:
    holdings:
      @attributes:
        islocal: N