Long-term outcomes after pre-emptive liver transplantation in primary hyperoxaluria type 1.

Background: Primary hyperoxaluria type 1 (PH1) is an autosomal recessive disease caused by the liver defect of oxalate metabolism, which leads to kidney failure and systemic manifestations. Until recently, liver transplantation was the only definitive treatment. The timing of liver transplantation c...

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Publicado en:Pediatric Nephrology Vol. 38; no. 6; pp. 1811 - 1821
Autores principales: Shasha-Lavsky, Hadas, Avni, Aviv, Paz, Ziv, Kalfon, Limor, Dror, Amiel A., Yakir, Orly, Zaccai, Tzipora Falik, Weissman, Irith
Formato: research tables/charts Journal Article
Publicado: Springer Nature Jun2023
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jun2023
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00467-022-05803-y
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        atl: Long-term outcomes after pre-emptive liver transplantation in primary hyperoxaluria type 1.
      aug:
        au:
          Shasha-Lavsky, Hadas
          Avni, Aviv
          Paz, Ziv
          Kalfon, Limor
          Dror, Amiel A.
          Yakir, Orly
          Zaccai, Tzipora Falik
          Weissman, Irith
        affil: Pediatric Nephrology Unit, Galilee Medical Center, Nahariya, Israel
      sug:
        subj:
          Liver Transplantation
          Carbohydrate Metabolism, Inborn Errors Surgery
          Treatment Outcomes
          Renal Insufficiency, Chronic Prevention and Control
          Early Intervention
          Oxalic Acids Urine
          Human
          Retrospective Design
          Conservative Treatment
          Comparative Studies
          Descriptive Statistics
          Glomerular Filtration Rate
          Renal Replacement Therapy
          Renal Insufficiency, Chronic Therapy
          Renal Insufficiency, Chronic Mortality
          Child
          Adolescence
          Adult
          Child: 6-12 years
          Adolescent: 13-18 years
          Adult: 19-44 years
      ab: Background: Primary hyperoxaluria type 1 (PH1) is an autosomal recessive disease caused by the liver defect of oxalate metabolism, which leads to kidney failure and systemic manifestations. Until recently, liver transplantation was the only definitive treatment. The timing of liver transplantation can be early, while kidney function is still normal (pre-emptive liver transplantation—PLT), or when the patient reaches stage 5 chronic kidney disease (CKD) and needs combined liver-kidney transplantation. We aimed to determine the long-term kidney outcomes of PLT in PH1 patients. Methods: A retrospective single-center study of PH1 patients who were followed in our center between 1997 and 2017. We compared the kidney outcomes of patients who underwent PLT to those who presented with preserved kidney function and did not undergo PLT. Results: Out of 36 PH1 patients, 18 patients were eligible for PLT (eGFR > 40 mL/min/1.73 m2 at the time of diagnosis). Seven patients underwent PLT (PLT group), while 11 continued conservative treatments (PLTn group). In the PLT group, the median eGFR at the time of PLT and at the end of the follow-up period (14–20 years) was 72 (range 50–89) and 104 (range 86–108) mL/min/1.73 m2, respectively, and no patient died or reached stage 5 CKD. In the PLTn group, eight patients (72.7%) reached stage 5 CKD (median time to kidney replacement therapy was 11 years), and two patients died from disease complications (18.2%). Conclusions: Pre-emptive liver transplantation preserved kidney function in patients with PH1 in our cohort. Early intervention can prevent kidney failure and systemic oxalosis in PH1.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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