Fucosyltransferase 5 Promotes the Proliferative and Migratory Properties of Intrahepatic Cholangiocarcinoma Cells via Regulating Protein Glycosylation Profiles.

Background: The incidence of intrahepatic cholangiocarcinoma (ICC) is increasing globally, and its prognosis has not improved substantially in recent years. Understanding the pathogenesis of ICC may provide a theoretical basis for its treatment. In this study, we investigated the effects and underly...

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Published in:Clinical Medicine Insights: Oncology pp. 1 - 11
Main Authors: Guo, Jingheng, Cheng, Qian, Li, Yongjian, Tian, Lingyu, Ma, Delin, Li, Zhao, Gao, Jie, Zhu, Jiye
Format: pictorial research tables/charts Journal Article
Published: Sage Publications Inc. 7/4/2023
Online Access:View this record in EBSCOhost
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      jtl: Clinical Medicine Insights: Oncology
      issn: 11795549
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      dt: 7/4/2023
      pid: 344
      pub: Sage Publications Inc.
      place: Thousand Oaks, California
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        164722086
        164722086
        164722086
        10.1177/11795549231181189
        164722086
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        atl: Fucosyltransferase 5 Promotes the Proliferative and Migratory Properties of Intrahepatic Cholangiocarcinoma Cells via Regulating Protein Glycosylation Profiles.
      aug:
        au:
          Guo, Jingheng
          Cheng, Qian
          Li, Yongjian
          Tian, Lingyu
          Ma, Delin
          Li, Zhao
          Gao, Jie
          Zhu, Jiye
        affil: Department of Hepatobiliary Surgery, Peking University People's Hospital, Beijing, China
      sug:
        subj:
          Transferases Metabolism
          Proteins Metabolism
          Glycosylation
          Cell Proliferation
          Cholangiocarcinoma
          Cell Movement
          Human
          Immunohistochemistry
          Mass Spectrometry
          Reverse Transcriptase Polymerase Chain Reaction
          Cell Culture Techniques
          Cell Viability
          Blotting, Western
          Descriptive Statistics
          T-Tests
          Data Analysis Software
      ab: Background: The incidence of intrahepatic cholangiocarcinoma (ICC) is increasing globally, and its prognosis has not improved substantially in recent years. Understanding the pathogenesis of ICC may provide a theoretical basis for its treatment. In this study, we investigated the effects and underlying mechanisms of fucosyltransferase 5 (FUT5) on the malignant progression of ICC. Methods: FUT5 expression in ICC samples and adjacent nontumor tissues was compared using quantitative real-time polymerase chain reaction and immunohistochemical assays. We performed cell counting kit-8, colony formation, and migration assays to determine whether FUT5 influenced the proliferation and mobility of ICC cells. Finally, mass spectrometry was performed to identify the glycoproteins regulated by FUT5. Results: FUT5 mRNA was significantly upregulated in most ICC samples compared with corresponding adjacent nontumor tissues. The ectopic expression of FUT5 promoted the proliferation and migration of ICC cells, whereas FUT5 knockdown significantly suppressed these cellular properties. Mechanistically, we demonstrated that FUT5 is essential for the synthesis and glycosylation of several proteins, including versican, β3 integrin, and cystatin 7, which may serve key roles in the precancer effects of FUT5. Conclusions: FUT5 is upregulated in ICC and promotes ICC development by promoting glycosylation of several proteins. Therefore, FUT5 may serve as a therapeutic target for the treatment of ICC.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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