Interrelationships among growth hormone, thyroid function, and endocrine-disrupting chemicals on the susceptibility to attention-deficit/hyperactivity disorder.

Abnormal growth hormones and thyroid function may be linked to pathophysiology of attention-deficit/hyperactivity disorder (ADHD). Phthalates and bisphenol-A (BPA), two endocrine-disrupting chemicals (EDCs), may affect the human endocrine system. In this study, we aimed to perform a comprehensive in...

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Publicado en:European Child & Adolescent Psychiatry Vol. 32; no. 8; pp. 1391 - 1402
Autores principales: Wang, Liang-Jen, Huang, Ying-Hua, Chou, Wen-Jiun, Lee, Sheng-Yu, Chang, Hsin-Yu, Chen, Chih-Cheng, Chao, How-Ran
Formato: research tables/charts Journal Article
Publicado: Springer Nature Aug2023
Acceso en línea:Ver este registro en EBSCOhost
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00787-021-01886-4
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        atl: Interrelationships among growth hormone, thyroid function, and endocrine-disrupting chemicals on the susceptibility to attention-deficit/hyperactivity disorder.
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        au:
          Wang, Liang-Jen
          Huang, Ying-Hua
          Chou, Wen-Jiun
          Lee, Sheng-Yu
          Chang, Hsin-Yu
          Chen, Chih-Cheng
          Chao, How-Ran
        affil: Department of Child and Adolescent Psychiatry, Kaohsiung Chang Gung Memorial Hospital and Chang Gung University College of Medicine, Kaohsiung, Taiwan
      sug:
        subj:
          Attention Deficit Hyperactivity Disorder Physiopathology
          Human Growth Hormone Blood
          Thyroid Hormones Blood
          Benzhydryl Compounds Urine
          Plasticizers Analysis
          Attention Deficit Hyperactivity Disorder Risk Factors
          Plasticizers Adverse Effects
          Benzhydryl Compounds Adverse Effects
          Risk Assessment
          Human
          Case Control Studies
          Child
          Somatomedins Blood
          Thyrotropin Blood
          Triiodothyronine Blood
          Thyroxine Blood
          Plasticizers
          Connective Tissue Growth Factor Blood
          Carrier Proteins Blood
          Urinalysis
          Attention Deficit Hyperactivity Disorder Symptoms
          Neuropsychological Tests
          Descriptive Statistics
          Female
          Male
          Funding Source
          Child: 6-12 years
          Female
          Male
      ab: Abnormal growth hormones and thyroid function may be linked to pathophysiology of attention-deficit/hyperactivity disorder (ADHD). Phthalates and bisphenol-A (BPA), two endocrine-disrupting chemicals (EDCs), may affect the human endocrine system. In this study, we aimed to perform a comprehensive investigation of whether growth hormone, thyroid function, and EDCs exhibited differential levels between ADHD patients and healthy controls. In total, 144 children with ADHD and 70 healthy control subjects were enrolled. Their endocrine systems were evaluated using the serum levels of insulin-like growth factor-1 (IGF-1), IGF-binding protein-3 (IGFBP-3), thyroid-stimulating hormone (TSH), triiodothyronine (T3), thyroxine (T4), and Free T4. The urinary levels of EDCs, including monoethyl phthalate (MEP), mono-methyl phthalate (MMP), monoethylhexyl phthalate (MEHP), mono-n-butyl phthalate (MnBP), monobenzyl phthalate (MBzP), and BPA, were also examined. Patients with ADHD had lower IGF-1 levels than healthy controls (p = 0.003), but we observed no significant difference in IGFBP-3, TSH, T3, T4, or Free T4. Compared to the control group, patients with ADHD demonstrated higher MEHP levels (p = 0.043), MnBP (p = 0.033), and MBzP (p = 0.040). Furthermore, MEHP levels (p < 0.001) and BPA levels (p = 0.041) were negatively correlated with IGF-1 levels, while IGF-1 levels were negatively correlated with principal components consisting of ADHD clinical symptoms and neuropsychological performance variables. We suggest that MEHP exposure may be associated with decreased serum levels of IGF-1 and increased risk of ADHD. The mechanism underlying this association may be important for protecting children from environmental chemicals that adversely affect neurodevelopment.
      pubtype: Academic Journal
      doctype:
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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