The beneficial effects of vortioxetine on BDNF, CREB, S100B, β amyloid, and glutamate NR2b receptors in chronic unpredictable mild stress model of depression.

Background: Depression, one of the most significant mental disorders, is still poorly understood in terms of its pathogenetic mechanisms despite its well-recognized association with stress. Objectives: The current study's goal was to ascertain how the novel antidepressant drug vortioxetine (VOR) aff...

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Publicado en:Psychopharmacology Vol. 240; no. 12; pp. 2499 - 2514
Autores principales: Ünal, Gülin Özdamar, Erkılınç, Gamze, Öztürk, Kuyaş Hekimler, Doguç, Duygu Kumbul, Özmen, Özlem
Formato: Journal Article
Publicado: Springer Nature Dec2023
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Dec2023
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      pub: Springer Nature
      place: New York, New York
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        169846416
        10.1007/s00213-023-06445-0
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        atl: The beneficial effects of vortioxetine on BDNF, CREB, S100B, β amyloid, and glutamate NR2b receptors in chronic unpredictable mild stress model of depression.
      aug:
        au:
          Ünal, Gülin Özdamar
          Erkılınç, Gamze
          Öztürk, Kuyaş Hekimler
          Doguç, Duygu Kumbul
          Özmen, Özlem
        affil: https://ror.org/04fjtte88 Faculty of Medicine, Department of Psychiatry, Suleyman Demirel University, Isparta, Turkey
      sug:
      ab: Background: Depression, one of the most significant mental disorders, is still poorly understood in terms of its pathogenetic mechanisms despite its well-recognized association with stress. Objectives: The current study's goal was to ascertain how the novel antidepressant drug vortioxetine (VOR) affected the BDNF (brain-derived neurotrophic factor), S100, amyloid β (Aβ), CREB (cAMP response element-binding protein), and NR2B, as well as its impact on depression-like behaviors, and tissue damage in an experimental rodent model of depression caused by chronic unpredictable stress. Methods: We employed twenty-eight Wistar albino male rats, and we randomly divided them into four groups, each consisting of 7 rats: control, CUMS (chronic unpredictable mild stress), CUMS+vortioxetine (CUMS+VOR), and CUMS+fluoxetine (CUMS+FLU). Sucrose preference and forced swimming tests (SPT and FST, respectively), PCR, ELISA, and histopathological and immunohistochemical evaluation were made on brains. Results: The behaviors of reduced immobility in the FST and increased sucrose preference were observed in the CUMS group and they improved in the groups treated with VOR and FLU. Compared with the control group, the group exposed to CUMS showed increased Aβ and decreased BDNF, CREB, and S-100 expressions, as well as neuronal degeneration (p<0.001). VOR and FLU treatment ameliorate the findings. Conclusions: This study demonstrated significant ameliorative effects of VOR in an experimental model of chronic unpredictable depression to reduce brain tissue damage and depression-like behaviors in rats.
      pubtype: Academic Journal
      doctype: Journal Article
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    language: English
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