Differences in whole-brain metabolism are associated with the expression of genes related to neurovascular unit integrity and synaptic plasticity in temporal lobe epilepsy.

Purpose: Temporal lobe epilepsy (TLE) is a common, polygenic epilepsy syndrome that involves glucose hypometabolism in the epileptogenic zone. However, the transcriptional and cellular signatures underlying the metabolism in TLE remain unclear. Methods: In this retrospective study, 2-[18F]-fluoro-2-...

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Publicado en:European Journal of Nuclear Medicine & Molecular Imaging Vol. 51; no. 1; pp. 168 - 180
Autores principales: Xiao, Ling, Tang, Yongxiang, Deng, Chijun, Li, Jian, Li, Rong, Zhu, Haoyue, Guo, Danni, Yang, Zhiquan, Long, Hongyu, Feng, Li, Hu, Shuo
Formato: Journal Article
Publicado: Springer Nature Dec2023
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Dec2023
      vid: 51
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      pub: Springer Nature
      place: New York, New York
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        171937284
        10.1007/s00259-023-06433-8
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        atl: Differences in whole-brain metabolism are associated with the expression of genes related to neurovascular unit integrity and synaptic plasticity in temporal lobe epilepsy.
      aug:
        au:
          Xiao, Ling
          Tang, Yongxiang
          Deng, Chijun
          Li, Jian
          Li, Rong
          Zhu, Haoyue
          Guo, Danni
          Yang, Zhiquan
          Long, Hongyu
          Feng, Li
          Hu, Shuo
        affil: Department of Nuclear Medicine, Xiangya Hospital, Central South University, 87 Xiangya Rd, 410008, Changsha, Hunan, China
      sug:
      ab: Purpose: Temporal lobe epilepsy (TLE) is a common, polygenic epilepsy syndrome that involves glucose hypometabolism in the epileptogenic zone. However, the transcriptional and cellular signatures underlying the metabolism in TLE remain unclear. Methods: In this retrospective study, 2-[18F]-fluoro-2-deoxy-D-glucose ([18F]FDG) positron emission tomography (PET) scans of TLE patients (n = 104) who underwent anterior temporal lobectomy were consecutively collected between 2016 and 2021. The transcriptional profiles of TLE risk genes across the brain were identified by the gene expression analyses from six TLE patients and twelve postmortem donors (six from the Allen Human Brain Atlas). Integrating the neuroimaging and transcriptomic data, we examined the relationship between the expression of TLE-associated genes and metabolic alterations in TLE. Furthermore, we performed functional enrichment analyses of the genes with higher weight in partial least squares regression using Metascape. Results: A total of 104 patients with TLE (mean age 29 ± 9 years, 50% male) and 30 healthy controls (HCs) (mean age 31 ± 6 years, 53% male) were enrolled. Compared to that of HCs, patients with TLE showed hypometabolism in the temporal lobes and adjacent structures but hypermetabolism in the thalamus and basal ganglia. The cortical map of inter-group differences in cerebral metabolism was spatially correlated with the expression of a weighted combination of genes enriched in ontology terms and pathways related to neurovascular unit (NVU) integrity and synaptic plasticity. Discussion: Our findings, combined with the analysis of neuroimaging and transcriptional data, suggest that genes related to NVU integrity and synaptic plasticity may drive alterations to brain metabolism that mediate the genetic risk of TLE.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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