Toxic effects of Aroclor 1254 on rat liver and modifying roles of selenium.

Polychlorinated biphenyls (PCBs) were used in different industrial areas and banned due to their high toxicity. Aroclor 1254 (A1254), commercial PCB congener, accumulates in environment leading to high human exposure. A1254 may cause hepatotoxicity, metabolic and endocrine disorders. In our study, 3...

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Publicado en:International Journal of Environmental Health Research Vol. 33; no. 12; pp. 1289 - 1305
Autores principales: Balcı Özyurt, Aylin, Erkekoğlu, Pınar, Zeybek, Naciye Dilara, Aşcı, Ali, Yaman, Ünzile, Oflaz, Ofcan, Kızılgün, Murat, İşcan, Evin, Batur, Tuğçe, Öztürk, Mehmet, Koçer-Gümüşel, Belma
Formato: pictorial research tables/charts Journal Article
Publicado: Taylor & Francis Ltd Dec2023
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Dec2023
      vid: 33
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      pub: Taylor & Francis Ltd
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        10.1080/09603123.2023.2223470
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        atl: Toxic effects of Aroclor 1254 on rat liver and modifying roles of selenium.
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        au:
          Balcı Özyurt, Aylin
          Erkekoğlu, Pınar
          Zeybek, Naciye Dilara
          Aşcı, Ali
          Yaman, Ünzile
          Oflaz, Ofcan
          Kızılgün, Murat
          İşcan, Evin
          Batur, Tuğçe
          Öztürk, Mehmet
          Koçer-Gümüşel, Belma
        affil: Department of Toxicology, Faculty of Pharmacy, Hacettepe University, Ankara, Turkey
      sug:
        subj:
          Hepatotoxicity Drug Therapy
          Liver Drug Effects
          Selenium Therapeutic Use
          Polychlorinated Biphenyls Poisoning
          Environmental Pollutants Poisoning
          Models, Biological
          Animal Studies
          Rats
          Thyroid Antagonists Adverse Effects
          Polychlorinated Biphenyls Administration and Dosage
          Polychlorinated Biphenyls Adverse Effects
          Funding Source
      ab: Polychlorinated biphenyls (PCBs) were used in different industrial areas and banned due to their high toxicity. Aroclor 1254 (A1254), commercial PCB congener, accumulates in environment leading to high human exposure. A1254 may cause hepatotoxicity, metabolic and endocrine disorders. In our study, 3-week-old male rats were separated into 6 groups: C (0.15 mg/kg Se in diet); SeS (1 mg/kg Se in diet); SeD (0.05 mg/kg Se in diet); A1254 receiving groups (A; ASeS; ASeD) were given 10 mg/kg/day A1254 orally for last 15 days of feeding period with control, SeD or SeS diet, respectively, for 5 weeks. Histopathology, oxidant/antioxidant balance, apoptosis and cell cycle proteins (p53, p21) in liver were evaluated. Our results suggest that A1254 leads to changes in histology, oxidative stress and apoptosis. Selenium deficiency augments oxidative stress and apoptosis while selenium supplementation is partially protective. More mechanistic in vivo experiments are necessary for evaluation of hepatotoxicity of PCBs.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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