Comparison of Pulmonary and Extrapulmonary Models of Sepsis-Associated Acute Lung Injury.

To compare different rat models of sepsis at different time points, based on pulmonary or extrapulmonary injury mechanisms, to identify a model which is more stable and reproducible to cause sepsis-associated acute lung injury (ALI). Adult male Sprague-Dawley rats were subjected to (1) cecal ligatio...

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Publicado en:Physiological Research Vol. 72; no. 6; pp. 741 - 753
Autores principales: Gengbiao ZHOU, Dongping XIE, Rongrong FAN, Zihong YANG, Jiongdong DU, Shutao MAI, Liying XIE, Qin WANG, Tong MAI, Yun HAN, Fang LAI
Formato: Journal Article
Publicado: Institute of Physiology, Academy of Sciences of the Czech Republic Dec2023
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Dec2023
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      pub: Institute of Physiology, Academy of Sciences of the Czech Republic
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        10.33549/physiolres.935123
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        atl: Comparison of Pulmonary and Extrapulmonary Models of Sepsis-Associated Acute Lung Injury.
      aug:
        au:
          Gengbiao ZHOU
          Dongping XIE
          Rongrong FAN
          Zihong YANG
          Jiongdong DU
          Shutao MAI
          Liying XIE
          Qin WANG
          Tong MAI
          Yun HAN
          Fang LAI
        affil: The Second Clinical College of Guangzhou University of Chinese Medicine, Guangzhou, Guangdong, China
      sug:
      ab: To compare different rat models of sepsis at different time points, based on pulmonary or extrapulmonary injury mechanisms, to identify a model which is more stable and reproducible to cause sepsis-associated acute lung injury (ALI). Adult male Sprague-Dawley rats were subjected to (1) cecal ligation and puncture (CLP) with single (CLP1 group) or two repeated through-and-through punctures (CLP2 group); (2) tail vein injection with lipopolysaccharide (LPS) of 10mg/kg (IVLPS10 group) or 20mg/kg (IV-LPS20 group); (3) intratracheal instillation with LPS of 10mg/kg (IT-LPS10 group) or 20mg/kg (IT-LPS20 group). Each of the model groups had a sham group. 7-day survival rates of each group were observed (n=15 for each group). Moreover, three time points were set for additional experimental studying in each model group: 4 hours, 24 hours and 48 hours after modeling (every time point, n=8 for each group). Rats were sacrificed to collect BALF and lung tissue samples at different time points for detection of IL-6, TNF-a, total protein concentration in BALF and MPO activity, HMGB1 protein expression in lung tissues, as well as the histopathological changes of lung tissues. More than 50 % of the rats died within 7 days in each model group, except for the IT-LPS10 group. In contrast, the mortality rates in the two IV-LPS groups as well as the IT-LPS20 group were significantly higher than that in ITLPS10 group. Rats received LPS by intratracheal instillation exhibited evident histopathological changes and inflammatory exudation in the lung, but there was no evidence of lung injury in CLP and IV-LPS groups. Rat model of intratracheal instillation with LPS proved to be a more stable and reproducible animal model to cause sepsis-associated ALI than the extrapulmonary models of sepsis.
      pubtype: Academic Journal
      doctype: Journal Article
      ougenre: Article
    language: English
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