Oxyresveratrol attenuates bone resorption by inhibiting the mitogen-activated protein kinase pathway in ovariectomized rats.

Background: Bone is continuously produced by osteoblasts and resorbed by osteoclasts to maintain homeostasis. Impaired bone resorption by osteoclasts causes bone diseases such as osteoporosis and arthritis. Most pharmacological treatment of osteoporosis focuses on inhibiting osteoclast differentiati...

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Publicado en:Nutrition & Metabolism Vol. 21; no. 1; pp. 1 - 14
Autores principales: Lee, Yea-Jin, Ahn, Jin-Chul, Oh, Chung-Hun
Formato: diagnostic images pictorial research tables/charts Journal Article
Publicado: BioMed Central 1/19/2024
Acceso en línea:Ver este registro en EBSCOhost
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      jtl: Nutrition & Metabolism
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      dt: 1/19/2024
      vid: 21
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      pub: BioMed Central
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        10.1186/s12986-024-00781-4
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        atl: Oxyresveratrol attenuates bone resorption by inhibiting the mitogen-activated protein kinase pathway in ovariectomized rats.
      aug:
        au:
          Lee, Yea-Jin
          Ahn, Jin-Chul
          Oh, Chung-Hun
        affil: https://ror.org/058pdbn81 Department of Medicine, Dankook University, 31116, Cheonan-si, Republic of Korea
      sug:
        subj:
          Osteoporosis Prevention and Control
          Bone Resorption Drug Therapy
          Stilbenes Pharmacodynamics
          Polyphenols Pharmacodynamics
          Bone Resorption Drug Effects
          Signal Transduction Drug Effects
          Bone Density Drug Effects
          Osteoclasts Drug Effects
          Cell Differentiation Drug Effects
          Oophorectomy
          Animal Studies
          Rats
          Molecular Structure
          Macrophages Drug Effects
          Osteoblasts
          Models, Biological
          Staining and Labeling
          RNA, Messenger Analysis
          Gene Expression Drug Effects
          Pyridines Urine
          Tomography, X-Ray Computed Methods
          NF-kappa B Drug Effects
          Phosphorylation Drug Effects
          Extracellular Space Drug Effects
          Transcription Factors Drug Effects
          Peptide Hydrolases Drug Effects
          Funding Source
      ab: Background: Bone is continuously produced by osteoblasts and resorbed by osteoclasts to maintain homeostasis. Impaired bone resorption by osteoclasts causes bone diseases such as osteoporosis and arthritis. Most pharmacological treatment of osteoporosis focuses on inhibiting osteoclast differentiation, often to restore osteoclast/osteoclast balance. However, recent osteoporosis treatments have various side effects. According to a recent study, resveratrol, known as a stilbenoid family, is known to increase bone density, and the osteoclast inhibitory effect was confirmed using oxyresveratrol, a stilbenoid family. Here, we investigated the effect of oxyresveratrol on osteoclast differentiation and an ovariectomized mouse model. Methods: Mouse leukemia monocyte/macrophage cell line RAW 264.7 was treated with oxyresveratrol, and cell cytotoxicity was confirmed by measuring MTT assay. Tartrate-resistant acid phosphatase (TRAP), an enzyme marker for osteoclasts, was confirmed by staining. In addition, osteoclast differentiation markers and MAPK-related markers were confirmed at the mRNA level and protein expression. The effect of oxyresveratrol was confirmed using ovariectomized mice. Deoxypyridinoline (DPD) was measured using mouse urine and TRAP activity was observed using serum. Bone mineral density was also measured using Micro-CT. Results: The polyphenol oxyresveratrol inhibited receptor activator of nuclear factor kappa-Β ligand (RANKL)-induced osteoclast differentiation of RAW 264.7 cells. Furthermore, oxyresveratrol inhibited TRAP activity and actin-ring formation. Moreover, oxyresveratrol suppressed the phosphorylation of the RANKL-induced mitogen-activated protein kinases (MAPKs) p38, JNK, and ERK and significantly reduced the expression of bone differentiation markers (NFATc1, cathepsin K, and TRAP). Conclusion: Oxyresveratrol inhibits osteoclast differentiation via MAPK and increases bone density in ovariectomized rats, suggesting it has therapeutic potential for bone diseases such as osteoporosis. We confirmed the osteoporosis prevention effect of OR in Raw 264.7 cells, and future studies should confirm the effect of OR using rat bone marrow-derived cells.
      pubtype: Academic Journal
      doctype:
        diagnostic images
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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