Donepezil promotes skin flap survival through activation of the HIF‐1α/VEGF signalling pathway.

Flaps are mainly used to repair wounds in the clinical setting but can sometimes experience ischaemic necrosis postoperatively. This study investigated whether donepezil, an acetylcholinesterase inhibitor, can enhance the survival rate of flaps. We randomly allocated 36 rats into control, low‐dose (...

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Publicado en:Wound Repair & Regeneration Vol. 32; no. 4; pp. 500 - 511
Autores principales: Lin, Hang, Wang, Kaitao, Yang, Jialong, Wang, An, Deng, Jiapeng, Lin, Dingsheng
Formato: pictorial research tables/charts Journal Article
Publicado: Wiley-Blackwell Jul2024
Acceso en línea:Ver este registro en EBSCOhost
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      dt: Jul2024
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      pub: Wiley-Blackwell
      place: Malden, Massachusetts
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        atl: Donepezil promotes skin flap survival through activation of the HIF‐1α/VEGF signalling pathway.
      aug:
        au:
          Lin, Hang
          Wang, Kaitao
          Yang, Jialong
          Wang, An
          Deng, Jiapeng
          Lin, Dingsheng
        affil: Department of Hand and Plastic Surgery, The Second School of Medicine, The Second Affiliated Hospital and Yuying Children's Hospital, Wenzhou Medical University, Wenzhou Zhejiang, , China
      sug:
        subj:
          Donepezil Pharmacodynamics
          Surgical Flaps
          Skin Transplantation
          Vascular Endothelial Growth Factors Drug Effects
          Signal Transduction Drug Effects
          Health Promotion Methods
          Graft Survival Drug Effects
          Proteins Drug Effects
          Animal Studies
          Mice
          Random Assignment
          Postoperative Period
          Tissue Viability Evaluation
          Molecular Biology
          Apoptosis Drug Effects
          Autophagy Drug Effects
          Neovascularization, Physiologic Drug Effects
          Interleukins
          Descriptive Statistics
          Biochemical Phenomena
          Funding Source
      ab: Flaps are mainly used to repair wounds in the clinical setting but can sometimes experience ischaemic necrosis postoperatively. This study investigated whether donepezil, an acetylcholinesterase inhibitor, can enhance the survival rate of flaps. We randomly allocated 36 rats into control, low‐dose (3 mg/kg/day), and high‐dose (5 mg/kg/day) groups. On Postoperative day 7, we assessed flap viability and calculated the mean area of viable flap. After euthanizing the rats, we employed immunological and molecular biology techniques to examine the changes in flap tissue vascularization, apoptosis, autophagy, and inflammation. Donepezil enhanced the expression of hypoxia‐inducible factor and vascular endothelial growth factor to facilitate angiogenesis. In addition, it elevated the expression of LC3B, p62, and beclin to stimulate autophagy. Furthermore, it increased the expression of Bcl‐2 while reducing the expression of Bax, thus inhibiting apoptosis. Finally, it had anti‐inflammatory effects by reducing the levels of IL‐1β, IL‐6, and TNF‐α. The results suggest that donepezil can enhance the viability of randomly generated skin flaps by upregulating HIF‐1α/VEGF signalling pathway, facilitating vascularization, inducing autophagy, suppressing cell apoptosis, and mitigating inflammation within the flap tissue.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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