CXCL5 Promotes the Malignant Phenotype of Pancreatic Cancer and Is Associated With Immune Infiltration.

Background: The significance of CXCL5 in pancreatic cancer is unclear, although it has been implicated in the malignant process of many different types of cancer. Research on the impact of CXCL5 on immune cell infiltration and the malignant phenotype of pancreatic cancer is needed. This study aimed...

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Publicado en:Clinical Medicine Insights: Oncology pp. 1 - 14
Autores principales: Wang, Tao, Sheng, Jian, Wang, Xiaoguang, Zhu, Minyuan, Li, Shijun, Shen, Yiyu, Wu, Bin
Formato: pictorial research tables/charts Journal Article
Publicado: Sage Publications Inc. 8/28/2024
Acceso en línea:Ver este registro en EBSCOhost
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      dt: 8/28/2024
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      pub: Sage Publications Inc.
      place: Thousand Oaks, California
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        atl: CXCL5 Promotes the Malignant Phenotype of Pancreatic Cancer and Is Associated With Immune Infiltration.
      aug:
        au:
          Wang, Tao
          Sheng, Jian
          Wang, Xiaoguang
          Zhu, Minyuan
          Li, Shijun
          Shen, Yiyu
          Wu, Bin
        affil: Graduate School, Zhejiang Chinese Medical University, Hangzhou, China
      sug:
        subj:
          Pancreatic Neoplasms Familial and Genetic
          Phenotype
          Gene Expression Physiology
          Pancreatic Neoplasms Immunology
          Neoplasm Invasiveness
          Human
          In Vitro Studies
          Animal Studies
          Mice
          China
          Correlational Studies
          Funding Source
          Pancreatic Neoplasms Prognosis
          Specimen Handling Methods
          Pancreatic Neoplasms Pathology
          Reverse Transcriptase Polymerase Chain Reaction Methods
          Chemokines
          Blotting, Western
          Cell Line, Tumor
          Descriptive Statistics
          Cell Proliferation
          Cell Movement
          Apoptosis
          Spearman's Rank Correlation Coefficient
      ab: Background: The significance of CXCL5 in pancreatic cancer is unclear, although it has been implicated in the malignant process of many different types of cancer. Research on the impact of CXCL5 on immune cell infiltration and the malignant phenotype of pancreatic cancer is needed. This study aimed to examine the connection between CXCL5 expression and immune cell infiltration and the malignant phenotype of pancreatic cancer. Methods: Tissue samples and clinical information were collected from 90 patients with pancreatic cancer. Tumour tissues and adjacent tissues were made into a tissue microarray and stained for immunohistochemistry analysis. Reverse transcription-quantitative polymerase chain reaction (RT-qPCR) and Western blot analysis were performed to measure the expression level of CXCL5. CXCL5-overexpressing/CXCL5-knockdown cell lines were constructed via transfection for cytological experiments. CCK-8, cell apoptosis, cell cycle, cell invasion, and cell colony formation assays were used to detect the effect of CXCL5 on the malignant phenotype of pancreatic cancer cells. Finally, a mouse model of pancreatic cancer was constructed for in vivo verification. Results: Compared with control cells, pancreatic cancer cells overexpressing CXCL5 exhibited increased proliferation, migration, and invasion but decreased apoptosis. Conversely, knockdown of CXCL5 did not enhance the malignant phenotype of pancreatic cancer cells. Spearman correlation analysis indicated that there was a significant negative correlation between CXCL5 levels and the CD8 IRS. However, there was a significant positive correlation between FOXP3 IRS and CXCL5 levels. Conclusions: CXCL5 is highly expressed in pancreatic cancer and promotes the malignant phenotype of pancreatic cancer cells. CXCL5 is associated with immunosuppressive FOXP3 + T-cell infiltration, which facilitates the formation of an immunosuppressive microenvironment (with low CD8 + T-cell infiltration).
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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