EGCG suppressed activation of hepatic stellate cells by regulating the PLCE1/IP3/Ca2+ pathway.

(-)-Epigallocatechin-3-O-gallate (EGCG), one of the green tea catechins, exhibits significant antioxidant properties that play an essential role in various diseases. However, the functional role and underlying mechanism of EGCG in stimulating of hepatic stellate cells (HSCs) remain unexplored in tra...

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Publicado en:European Journal of Nutrition Vol. 63; no. 8; pp. 3255 - 3269
Autores principales: Lin, Ying, Zhang, Yan, Li, Yang, Xu, Qihan, Zhang, Yijie, Chen, Tingting, Wang, Jun, Li, Jierui, Gong, Jiacheng, Chen, Zhuoer, Yang, Qiaomu, Li, Xu
Formato: pictorial research tables/charts Journal Article
Publicado: Springer Nature Dec2024
Acceso en línea:Ver este registro en EBSCOhost
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      jtl: European Journal of Nutrition
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      dt: Dec2024
      vid: 63
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      pub: Springer Nature
      place: New York, New York
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        10.1007/s00394-024-03504-w
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        atl: EGCG suppressed activation of hepatic stellate cells by regulating the PLCE1/IP3/Ca2+ pathway.
      aug:
        au:
          Lin, Ying
          Zhang, Yan
          Li, Yang
          Xu, Qihan
          Zhang, Yijie
          Chen, Tingting
          Wang, Jun
          Li, Jierui
          Gong, Jiacheng
          Chen, Zhuoer
          Yang, Qiaomu
          Li, Xu
        affil: Department of Emergency Medicine, Nanfang Hospital, Southern Medical University, No. 1838, North of Guangzhou Avenue, 510515, Guangzhou City, Guangdong Province, China
      sug:
        subj:
          Liver Cirrhosis Drug Therapy
          Flavonoids Pharmacodynamics
          Green Tea Pharmacodynamics
          Cell Line Drug Effects
          Phospholipases Metabolism
          Calcium Metabolism
          Signal Transduction Drug Effects
          Hydrocarbons, Chlorinated Adverse Effects
          Administration, Oral
          Gene Expression Profiling
          Antioxidants
          Green Tea Administration and Dosage
          Animal Studies
          China
          Mice
          In Vitro Studies
          Sequence Analysis
          Inositol Phosphates Metabolism
          Cell Proliferation
          Cell Movement
          Reactive Oxygen Species
          Transcription Factors
          Descriptive Statistics
      ab: (-)-Epigallocatechin-3-O-gallate (EGCG), one of the green tea catechins, exhibits significant antioxidant properties that play an essential role in various diseases. However, the functional role and underlying mechanism of EGCG in stimulating of hepatic stellate cells (HSCs) remain unexplored in transcriptomics sequencing studies. The present study suggests that oral administration of EGCG at a dosage of 200 mg/kg/day for a duration of four weeks exhibits significant therapeutic potential in a murine model of liver fibrosis induced by CCl4. The activation of HSCs in vitro was dose-dependently inhibited by EGCG. The sequencing analysis data reveled that EGCG exerted a regulatory effect on the calcium signal in mouse HSCs, resulting in a decrease in calcium ion concentration. Further analysis revealed that EGCG inhibited the expression of phospholipase C epsilon-1 (PLCE1) and inositol 1, 4, 5-trisphosphate (IP3) in activated mouse HSCs. Additionally, EGCG contributes to the reduction the concentration of calcium ions by regulating PLCE1. After the knockdown of PLCE1, free calcium ion concentrations decreased, resulting in the inhibition of both cell proliferation and migration. Interestingly, the expression of PLCE1 and cytosolic calcium levels were regulated by reactive oxygen species(ROS). Furthermore, our findings suggest that ROS might inhibit the expression of PLCE1 by inhibiting TFEB, a transcription activator involved in the nuclear translocation process. Our study provided novel evidence regarding the regulatory effects of EGCG on activated HSCs (aHSCs) in mice by the calcium signaling pathway, emphasizing the crucial role of PLCE1 within the calcium signaling network of HSCs. The proposition was also made that PLCE1 holds promise as a novel therapeutic target for murine liver fibrosis.
      pubtype: Academic Journal
      doctype:
        pictorial
        research
        tables/charts
        Journal Article
      ougenre: Article
    language: English
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